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A previously healthy 53 yo woman was transferred to a receiving hospital in cardiogenicshock. Here was the ECG: There is sinus tachycardia. Referring to Figure-1 — this 53-year old woman who presented in extremis with cardiogenicshock and an initial pH = 6.9, This was sent by a reader. and K was normal.
She presented to an outside hospital after several days of malaise and feeling unwell. Sinus tachycardia has many potential causes. This is especially true for the elderly patient with sinus tachycardia. What is the cause of the sudden tachycardia? The VSR is what is causing the cardiogenicshock!
Author continued : STE in aVR is often due to left main coronary artery obstruction (OR 4.72), and is associated with in-hospital cardiovascular mortality (OR 5.58). Authors' commentary: Cardiogenicshock in the setting of severe aortic stenosis. Fundamentally, cardiogenicshock is an issue of decreased cardiac output.
The patient had a protracted hospitalization and did not survive. There is sinus tachycardia at ~100/minute. As often emphasized by Dr. Smith — sinus tachycardia is not a common finding with acute OMI unless something else is going on (ie, cardiogenicshock ). The initial ECG for Patient #2 also shows RBBB.
Category 2 : An increase in myocardial oxygen demand due to tachycardia, elevated ventricular afterload (BP or aortic stenosis), or increased wall stretch (admittedly this latter is more complicated) or a decrease in oxygen supply due to hypotension, anemia, hypoxia, or a combination of all of the above. This results in Type I MI.
An elderly man with sudden cardiogenicshock, diffuse ST depressions, and STE in aVR Literature 1. However, STE-aVR with multilead ST depression was associated with 31% in-hospital mortality compared with only 6.2% A slightly prolonged QTc ( although this is difficult to assess given the tachycardia ). Left main?
Figure B At this point, with the ECG changing from diffuse ST depression to widespread ST elevation and the patient presenting in cardiogenicshock, left main coronary artery (LMCA) occlusion is the likely diagnosis. He was later transferred back to his local hospital neurologically intact and without serious sequela.
The patient in today’s case presented in cardiogenicshock from proximal LAD occlusion, in conjunction with a subtotally stenosed LMCA. The patient was extubated on Day-3 of the hospital stay. The patient improved, and on Day-11 of the hospital stay — he was off inotropes and on a small dose of a ß-blocker.
There is sinus tachycardia (do not be fooled into thinking this is VT or another wide complex tachycardia!) This pattern is essentially always accompanied by cardiogenicshock and high rates of VT/VF arrest, etc. The patient arrived to the ED in cardiogenicshock but awake. in-hospital mortality was 18.8%
Assessment was severe sudden cardiogenicshock. 3 studied 416 patients hospitalized with COVID in China, of whom 82 had an initial cTn(I) above the upper reference limit. Association of Cardiac Injury With Mortality in Hospitalized Patients With COVID-19 in Wuhan, China. They recorded an EC G: New ST Elevation.
Why is the patient in shock? He was in profound cardiogenicshock. Both of these features make inferior + RV MI by far the most likely ( Pseudoanteroseptal MI is another name for this ) There is also sinus bradycardia and t he patient is in shock with hypotension. There is an obvious inferior STEMI, but what else?
Here is another proven left main occlusion in a young woman who presented with sudden pulmonary edema, had this ECG recorded, then arrested and was resuscitated after 30 minutes of CPR: This has sinus tachycardia with RBBB and LAFB, and STE in V2-V6 as well as I, aVL This pattern could just as easily be seen in LAD occlusion.
The Golden Hour post the Myocardial infarction (MI) It is a matter of great concern that 50% of individuals experiencing an acute heart attack pass away before reaching the hospital. The abnormal heart rhythms can further lead to death because of ventricular tachycardia and ventricular fibrillation.
The status of the patients chest pain at this time is unknown : EKG 1, 1300: There is sinus tachycardia and artifact of low and high frequency. However, there is also significant tachycardia , with heart rate of 116, and known hypoxia. Cardiology services were consulted at a PCI capable hospital. An EKG was immediately recorded.
1) as far as I can tell, there is very little data on amiodarone for this indication 2) amiodarone has beta blockade effects which could be deleterious in a patient with large anterior MI with pulmonary edema and at risk for cardiogenicshock (and she did go into shock. DOI: 10.1016/j.resuscitation.2025.110515
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