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Written by Jesse McLaren Two patients in their 70s presented to the ED with chestpain and RBBB. Patient 1 : a 75 year old called paramedics with one day of left shoulder pain which migrated to the central chest, which was worse with deep breaths. Ten days later the patient returned with worsening pleuritic chest.
A middle-aged patient with lung cancer had presented to clinic complaining of generalized malaise, cough, and chestpain. Symptoms other than chestpain (malaise, cough in a cancer patient) 2. I have always said that tachycardia should argue against acute MI unless there is cardiogenicshock or 2 simultaneous pathologies.
Taking a step back , remember that sinus tachycardia is less commonly seen in OMI (except in cases of impending cardiogenicshock). In patients with narrow QRS ( not this patient), this pattern is highly suggestive of acute pulmonary embolism. In PE, there is almost always some hypoxia without any pulmonary edema.
large ASD, partial anomalous pulmonary venous return, significant tricuspid regurgitation, carcinoid valvular disease, etc,) 2) Conditions causing pressure overload of the RV. Any cause of pulmonary hypertension. There is normal axis, normal R-wave progression in the precordial leads and no intraventricular conduction abnormalities.
PEARL # 2: In the absence of associated heart failure ( cardiogenicshock ) — sinus tachycardia is not a common finding in acute MI. Today’s patient presented to the ED not only with chestpain — but also with shortness of breath , therefore with a history potentially consistent with the diagnosis.
Notice I did not say "pulmonary embolism," because any form of severe acute right heart strain may produce this ECG. This includes, but is not limited to, PE, asthma/COPD exacerbation, hypoxic vasoconstriction from pneumonia, acute pulmonary hypertension exacerbation. Differences of Pulmonary Embolism T-waves from Wellens' T-waves: 1.
It was edited by Smith CASE : A 52-year-old male with a past medical history of hypertension and COPD summoned EMS with complaints of chestpain, weakness and nausea. The patient was transported to the CCU for further medical optimization where a pulmonary artery catheter was placed.
My answer: "This is classic for PE, but it can also be present in any hypoxia due pulmonary hypoxic vasoconstriction and resulting acute pulmonary hypertension and acute right heart strain. Tachycardia is unusual in ACS unless there is cardiogenicshock or a second simultaneous pathology. This is NOT Wellens.
Then the notes mention "cardiogenicshock" but without any reference to a cardiac echo or to a chest x-ray. Was there pulmonary edema? Now chestpain free. Cardiologist note says: "Elevated troponin explained by type II MI due to her shock." Not mentioned in physicians' notes.
A man in his 60's presented after 4 days of chestpain, with some increase of pain on the day of presentation. Exact pain history was difficult to ascertain. Case Continued 2 days later the patient became increasingly tachycardic, hypotensive, ashen, clammy (in cardiogenicshock) and had a new murmur.
His comments/questions are inserted below the ECG: A 50-something woman presented with 3 days of intermittent chestpain that became worse on the day of presentation, with diaphoresis and radiation to the left arm, as well as abdominal pain. This is her ECG: An obvious STEMI, but which artery?
When a person experiences a heart attack or myocardial infarction, they may feel chestpain and other symptoms in different parts of their body. So, how do you recognize a heart attack? Myocardial infarction Symptoms Recognizing the signs of a heart attack before seeking medical attention is vital.
A 69 year old woman with a history of hypertension presented to the emergency department by EMS for evaluation of chestpain and shortness of breath. She awoke in the morning with sharp chestpain which worsened throughout the morning. As her pain worsened, so did her dyspnea. This was written by Hans Helseth.
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