Cardiac Arrest Ischemia Pharmacology 
Dr. Smith's ECG Blog
OCTOBER 23, 2023
Edited by Bracey, Meyers, Grauer, and Smith A 50-something-year-old female with a history of an unknown personality disorder and alcohol use disorder arrived via EMS following cardiac arrest with return of spontaneous circulation. The described rhythm was an irregular, wide complex rhythm.
Dr. Smith's ECG Blog
APRIL 8, 2016
If there is polymorphic VT with a long QT on the baseline ECG, then generally we call that Torsades, but Non-Torsades Polymorphic VT can result from ischemia alone. If cardiac arrest from hypokalemia is imminent (i.e., As I indicated above, in our cardiac arrest case, after pushing 40 mEq, the K only went up to 4.2
DAIC
JANUARY 23, 2024
mg reduced the risk of cardiovascular death, MI or heart attack, ischemic stroke, or ischemia-driven coronary revascularization by 31% compared with placebo.34 Therapeutic potential of colchicine in cardiovascular medicine: a pharmacological review. This in turn leads to an overall reduction in IL-6 production and CRP concentration.12
Dr. Smith's ECG Blog
SEPTEMBER 7, 2020
If the rhythm converts and then reverts to tachycardia with either adenosine or electricity, Neither one of those modalities should be attempted until some longer acting pharmacological solution is given. WPW does not always have delta waves on the baseline ECG. This is called "Concealed conduction".
Dr. Smith's ECG Blog
NOVEMBER 28, 2022
Alternation in ST segment appearance ( or in the amount of ST elevation or depression ) — is often linked to ischemia. This may result from fluctuations in heart rate or in nervous system activity or from pharmacologic treatment. Repolarization Alternans — entails beat-to-beat variation in the ST segment and/or T wave.
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