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It is seldom done in pediatric age group. This is mainly to account for the individual variation in anatomical location of right ventricular outflow tract, the main location of electrophysiological abnormalities in Brugada syndrome. Opinion is divided on the need for electrophysiology study.
Pediatric and elderly patients were more predisposed to developing an arrhythmic event in the setting of fever [7]. She has not yet been seen by electrophysiology or had further genetic testing for Brugada syndrome. She has not had a heart catheterization or after this event so the presence or absence of CAD is still unknown.
Pediatric and elderly patients were more predisposed to developing an arrhythmic event in the setting of fever [7]. She has not yet been seen by electrophysiology or had further genetic testing for Brugada syndrome. She has not had a heart catheterization or after this event so the presence or absence of CAD is still unknown.
In this pediatric study, it was 71% successful and better than amiodarone. Admission and referral to electrophysiology is always indicated. Procainamide is another reasonable solution to the problem. It does not block the AV node but does slow phase 0 of depolarization, which will also frequently break the re-entrant cycle.
Circulation: Arrhythmia and Electrophysiology, Ahead of Print. BACKGROUND:Sudden cardiac death is the most common cause of death in childhood hypertrophic cardiomyopathy (HCM). Recently, 2 risk scores have been developed to estimate the 5-year risk of sudden cardiac death. males), with a mean follow-up of 8.65.5
Further history later: This patient personally has no further high risk features (syncope / presyncope), but her mother had sudden cardiacarrest in sleep. We repeated the ECG: Brugada pattern is mostly resolved. Follow up the next AM: Brugada pattern is resolved Below is what the electrophysiologist recommended.
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