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Bedside cardiac ultrasound showed moderately decreased LV function. CT of the chest showed no pulmonary embolism but bibasilar infiltrates. See this post: How a pause can cause cardiacarrest 2. She was intubated. The plan: 1. Place temporary pacemaker 3. Discontinue amiodarone, since it prolongs the QT 4.
The morphology of V2-V4 is very specific in my experience for acute right heart strain (which has many potential etiologies, but none more common and important in EM than acute pulmonary embolism). He had multiple cardiacarrests with ROSC regained each time. CT angiogram showed extensive saddle pulmonary embolism.
Bedside ultrasound showed no effusion and moderately decreased LV function, with B-lines of pulmonary edema. Could the dysrhythmias have been prevented? If cardiacarrest from hypokalemia is imminent (i.e., As I indicated above, in our cardiacarrest case, after pushing 40 mEq, the K only went up to 4.2
They felt that the asystolic arrest suggested a different etiology of cardiacarrest. With the severe acidosis and absence of ST elevation, they felt there was more likely to be a non-cardiac etiology of his presentation. There was no pulmonary embolism.
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