Remove Cardiac Arrest Remove Defibrillator Remove Sudden Cardiac Death
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Smidt Heart Institute Sudden Cardiac Arrest Expert Receives 2024 Distinguished Scientist Award

DAIC

Particularly, his contributions to the sudden cardiac arrest medical knowledge base have changed the way we think about this deadly condition that we might be able to prevent on a larger scale.” Although “sudden cardiac arrest” and “heart attack” are often mistaken to be the same, the conditions are quite different.

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Inequities in access and use of automated external defibrillators

Heart BMJ

Sudden cardiac death is one of the main causes of mortality in patients with known or occult cardiac disease and is potentially preventable by early resuscitation. 1 Underuse of AEDs is a multifaceted problem with the number of available devices in a given distance to a person with out-of-hospital cardiac arrest (OHCA).

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Case Report: Lacosamide unmasking SCN5A-associated Brugada syndrome in a young female with epilepsy

Frontiers in Cardiovascular Medicine

During the intravenous lacosamide infusion, the patient developed sudden cardiac arrest caused by ventricular arrhythmias necessitating resuscitation. Of note, the patient had a family history of sudden cardiac death. 2893C>T, p.Arg965Cys) in the SCN5A gene.

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ECG Blog #436 — Bigeminy or Alternans?

Ken Grauer, MD

He developed cardiac arrest shortly after the ECG in Figure-1 was recorded. C ASE C onclusion: As noted above — today's patient developed cardiac arrest shortly after arrival in the ED. Despite prolonged resuscitation with multiple defibrillation attempts — the patient could not be saved. =

Blog 160
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Flecainide Is Associated With a Lower Incidence of Arrhythmic Events in a Large Cohort of Patients With Catecholaminergic Polymorphic Ventricular Tachycardia

Circulation

The study period was defined as the period in which background therapy (ie, beta-blocker type [beta1-selective or nonselective]), left cardiac sympathetic denervation, and implantable cardioverter defibrillator treatment status, remained unchanged within individual patients and was divided into pre-flecainide and on-flecainide periods.

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Risk Prediction in Male Adolescents With Congenital Long QT Syndrome: Implications for Sex?Specific Risk Stratification in Potassium Channel?Mediated Long QT Syndrome

Journal of the American Heart Association

Anderson‐Gill modeling was performed for the end point of CE burden (total number of syncope, aborted cardiac arrest, and appropriate defibrillator shocks). The applicability of the CE prediction model was tested for the end point of the first LTE (excluding syncope and adding sudden cardiac death) using Cox modeling.

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A young F is hyperthermic, delirious, and dry: Fever-induced Brugada? Diphenhydramine toxicity? Tricyclic?

Dr. Smith's ECG Blog

Further history later: This patient personally has no further high risk features (syncope / presyncope), but her mother had sudden cardiac arrest in sleep. Twenty-one percent (18 of 88) had a family history of sudden cardiac death and 26.4% (14 of 53) carried a pathogenic SCN5A mutation.