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A 50-year-old woman with dextrocardia and D-transposition of the great arteries presented with a “RV defib lead impedance” remote-monitoring alert from her implantable cardioverter defibrillator (ICD). In 2012, she had a ventricular fibrillation cardiacarrest. An epicardial pacemaker was implanted.
She was found to be in ventricular fibrillation and was defibrillated 8 times without a single, even transient, conversion out of fibrillation. She was immediately intubated during continued compressions, then underwent a 9th defibrillation, which resulted in an organized rhythm at 42 minutes after initial arrest.
She was defibrillated successfully from ventricular fibrillation and developed a perfusing rhythm. Pressors were required, and the patient was transported to the cath lab with a door to balloon time of 60 minutes, where a proximal dominant RCA occlusion was opened and stented. She was intubated.
The patient had 2 ventricular fibrillation arrests during transport, but was immediately defibrillated both times, and was awake in the ED, when the following ECG was recorded: The ST elevation has mostly resolved on this ECG, and were it not for the arrest and the prehospital ECG, this would not be a slam dunk diagnosis.
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At cath, he immediately had incessant Torsades de Pointes requiring defibrillation 7 times and requiring placement of a transvenous pacer for overdrive pacing at a rate of 80. This was stented. If cardiacarrest from hypokalemia is imminent (i.e., He was given amiodarone and lidocaine load and drip and K and Mg drips.
In the ambulance during transport, the patient suddenly suffered VF arrest. He was defibrillated immediately and had return of normal mental status. Given the VF arrest during transport, however, they appropriately all agreed that cath should happen sooner rather than later. They took him almost immediately for catheterization.
After the second defibrillation the patient had an organized rhythm: Bradycardic escape/agonal rhythm, with large ST deviations. The patient was taken back to the cath lab, where 100% proximal in-stent rethrombosis was found and treated. It should have been shocked at least 10 seconds ago. This is diagnostic of re-occlusion.
He was sent back to the waiting room, where he suffered a VF arrest. Defibrillation was performed, and ROSC was achieved. Smith comment: The patient was lucky to have a cardiacarrest. Total proximal LAD occlusion was found and stented at angiography soon after the ECG above.
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