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This was interpreted by the treating clinicians as not showing any evidence of ischemia. Given the presentation, the cardiologist stented the vessel and the patient returned to the ICU for ongoing criticalcare. He was intubated in the field and sedated upon arrival at the hospital. Two subsequent troponins were down trending.
If there is polymorphic VT with a long QT on the baseline ECG, then generally we call that Torsades, but Non-Torsades Polymorphic VT can result from ischemia alone. If cardiacarrest from hypokalemia is imminent (i.e., As I indicated above, in our cardiacarrest case, after pushing 40 mEq, the K only went up to 4.2
It is critically important for all EM and criticalcare providers to have an intimate understanding of hyperkalemia and its ECG findings. Steve, what do you think of this ECG in this CardiacArrest Patient?" HyperKalemia with CardiacArrest. Is this just right bundle branch block?
The patient might be having cardiacischemia, but if he is, it is unstable angina or non-STEMI, or perhaps he has not YET pseudonormalized, so serial ECGs may be important. Patients who present with chest pain or cardiacarrest and have an ECG diagnostic of STEMI could have myocardial rupture. These patients may survive.
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