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Photo by Cedars-Sinai milla1cf Fri, 03/01/2024 - 08:25 March 1, 2024 — Two new studies by Cedars-Sinai investigators support using artificial intelligence (AI) to predict sudden cardiacarrest-a health emergency that in 90% of cases leads to death within minutes.
Sent by Magnus Nossen MD, written by Pendell Meyers A man in his 50s, previously healthy, developed acute chestpain. The primary care physician there evaluated this patient and deemed the chestpain to be due to gastrointestinal causes. The ECG was also interpreted as normal by the primary care physician.
The chestpain quickly subsided. Cardiacarrest was called and advanced life support was undertaken for this patient. The patient was given chest compressions while waiting for the cardiacarrest team to arrive. After about 90 seconds of chest compressions she awoke. Calcium level was normal.
However, he did not remember much from the day of the arrest. He did not remember whether he had experienced any chestpain. Within a few days, the patient was extubated and was neurologically intact. At his family's request, he was transferred to a hospital closer to his home to continue care. He was admitted to cardiology.
But cardiacarrest is a period of near zero flow in the coronary arteries and causes SEVERE ischemia. After cardiacarrest, I ALWAYS wait 15 minutes after an ECG like this and record another. Just as important is pretest probability: did the patient report chestpain prior to collapse?
Written by Jesse McLaren Two patients in their 70s presented to the ED with chestpain and RBBB. Patient 1 : a 75 year old called paramedics with one day of left shoulder pain which migrated to the central chest, which was worse with deep breaths. Do either, both, or neither have occlusion MI? Vitals were normal.
Let me tell you about her hospitalization, discharged 1 day prior, but it was at another hospital (I wish I had the ECG from that hospitalization): The patient is 40 years old and presented to another hospital with chestpain and SOB. She had been sitting doing work when she experienced "waves of chest tightness". Sats were 88%.
Introduction Sudden cardiacarrest is a major cause of morbidity and mortality worldwide and remains a major public health problem for which better non-invasive prediction tools are needed. The individual relationship between fatal arrhythmias and cardiac function abnormalities in predicting cardiac death risk has rarely been explored.
A woman in her 50s with dyspnea and bradycardia A patient with cardiacarrest, ROSC, and right bundle branch block (RBBB). HyperKalemia with CardiacArrest. Steve, what do you think of this ECG in this CardiacArrest Patient?" Is this just right bundle branch block? What is it?
A male in late middle age with a history of RCA stent 8 years prior complained of chestpain. Here are three more dramatic cases that illustrate RBBB + LAFB Case 1 of cardiacarrest with unrecognized STEMI, died. EMS recorded the following ECG: What do you see?
Case submitted by Rachel Plate MD, written by Pendell Meyers A man in his 70s presented with chestpain which had started acutely at rest and has lasted for 2 hours. The pain was still ongoing at arrival. His initial troponin T was 15 ng/L (only two hours since pain onset). He stated it was similar to prior heart attacks.
ObjectiveAlthough the association between admission glucose (AG) and major adverse cardiac events (MACE) is well-documented, its relationship with 30-day MACE in patients presenting with cardiacchestpain remains unclarified.
There is no way to tell the difference between GI etiology of chestpain and MI. Such T-waves are almost always reciprocal to ischemia in the region of aVL (although aVL looks n ormal here) , and in a patient with chestpain are nearly diagnostic of ischemia. Could this have been avoided? Lesson : 1.
A patient had a cardiacarrest with ventricular fibrillation and was successfully defibrillated. Coronary Angiography after CardiacArrest without ST-Segment Elevation. N Engl J Med [Internet] 2019;Available from: [link] Should all patients with shockable arrest be taken to angiography regardless of STEMI or No STEMI?
A 20-something male drank heavily of ethanol and used cocaine, then was involved in a stressful verbal altercation, at which time he developed chestpain. See this post and associated case reports: Cardiacarrest, severe acidosis, and a bizarre ECG The patient was admitted and ruled out for acute MI by serial troponins.
This is another case written by Pendell Meyers (who is helping to edit the blog and has many great recent posts) Case A 45 year old man was driving to work when he experienced acute onset sharp left sided chestpain with paresthesias of the left arm. A repeat ECG was recorded with pain 2/10: Not much change.
The patient presented due to chestpain that was typical in nature, retrosternal and radiating to the left arm and neck. He denied any exertional chestpain. It is unclear if the patient was pain free at this time. He has a medical hx notable for hypertension, hyperlipidemia and previous tobacco use disorder.
He complained of severe chestpain and was extremely agitated, so much so that he was throwing chairs in triage. Some time later, reperfusion T-waves developed (analogous to Wellens' waves): Case 3 Here is a case of a 30-something otherwise healthy male with chestpain: There is neither an S-wave nor J-wave in lead V3.
The patient presented with chestpain. I was taught that the tell-tale sign of ischemia vs an electrical abnormality was in the hx, i.e. chestpain for the ischemia and potential syncope for brugada. Only 5-18% of ED patients with chestpain have a myocardial infarction of any kind. Is it Brugada pattern?
This 80 year old with a history of CABG had a cardiacarrest. He did not state he had chestpain, but, then again, he couldn't remember anything. He was resuscitated after fairly prolonged down time, but regained consciousness, though he was confused. There is concordant ST elevation in all inferior leads.
Written by Pendell Meyers and Peter Brooks MD A man in his 30s with no known past medical history was reported to suddenly experience chestpain and shortness of breath at home in front of his family. He had multiple cardiacarrests with ROSC regained each time. Submitted by a Med Student, with Great Commentary on Bias!
The patient was diagnosed with esophageal reflux and was being discharged by the nurse when he had a cardiacarrest. Formula : There is not enough ST elevation in V2-V4 to be applying the LAD/early repol formula, but if it is applied, one gets 1.5 The formula results in 23.43, just above the 23.4 He was defibrillated.
Patients may feel a fluttering in the chest, chestpain, shortness of breath and dizziness or lightheadedness as a result. If VT is not treated, it can lead to cardiacarrest, which is when the heart stops beating. In fact, VT is the most common cause of sudden cardiacarrest.
A small proportion of patients with STEMI treated via primary PCI experienced late ventricular tachycardia (VT) or ventricular fibrillation (VF), occurring one or more days following the procedure, but late VT or VF with cardiacarrest occurred rarely, especially among patients with uncomplicated STEMI, according to a study published in JAMA Network (..)
Two recent interventions have proven in randomized trials to improve neurologic survival in cardiacarrest: 1) the combination of the ResQPod and the ResQPump (suction device for compression-decompression CPR -- Lancet 2011 ) and 2) Dual Sequential defibrillation.
Because the patient had no chestpain or shortness of breath, they were initially diagnosed as gastroenteritis. But because the patient had no chestpain or shortness of breath, it was not deemed to be from ACS. Potassium was normal. Cardiology did not think it was "STEMI", but repeated the troponin. Take home 1.
A 60-something woman presented after a witnessed cardiacarrest. This is commonly found after epinephrine for cardiacarrest, but could have been pre-existing and a possible contributing factor to cardiacarrest. A recent similar case: A 40-something with chestpain. Is this inferior MI?
Submitted and written by Quinton Nannet, MD, peer reviewed by Meyers, Grauer, Smith A woman in her 70s recently diagnosed with COVID was brought in by EMS after she experienced acute onset sharp midsternal chestpain without radiation or dyspnea. She felt nauseous and lightheaded with no neurologic deficits.
One hour later (labs not yet returned), here is the ECG recorded just after the team noticed a sudden wide complex with precipitous decompensation, just before cardiacarrest: Bizarre, Brady, and Broad (wide QRS). Given the absence of chestpain — cardiac contusion is also unlikely.
Shortly after arrival in the ED ( E mergency D epartment ) — she suffered a cardiacarrest. BUT — Cardiac catheterization done a little later did not reveal any significant stenosis. Figure-1: The initial ECG in today's case — obtained after successful resuscitation from cardiacarrest. ( No CP ( C hest P ain ).
A 56 yo f with h/o HTN and hypercholesterolemia called EMS from home after onset of L chestpain radiating to the left arm. Before EMS arrived, she had "seizure activity" and became unresponsive. She was defibrillated successfully from ventricular fibrillation and developed a perfusing rhythm. She was intubated.
BACKGROUND:There is no specific treatment for sudden cardiacarrest (SCA) manifesting as pulseless electric activity (PEA) and survival rates are low; unlike ventricular fibrillation (VF), which is treatable by defibrillation. Circulation: Arrhythmia and Electrophysiology, Ahead of Print.
He did not have chestpain. Polymorphic Ventricular Tachycardia Long QT Syndrome with Continuously Recurrent Polymorphic VT: Management CardiacArrest. Chestpain in high risk patient. Here is his triage ECG: What do you think? What does the ECG show? Also see the bizarre Bigeminy. Is it STEMI?
CardiacArrest or Sudden Death: Cardiomegaly increases the risk of life-threatening arrhythmias, which can cause sudden cardiacarrest. Blood Clots: An enlarged heart is more prone to developing blood clots, which can lead to stroke or pulmonary embolism.
The ECG in Figure-1 — was obtained from a middle-aged man who presented to the ED ( E mergency D epartment ) in cardiacarrest. C ASE C onclusion: As noted above — the middle-aged man in today's case presented to the ED in cardiacarrest. In view of this history — How would YOU interpret the ECG in Figure-1 ?
He was at the gym when he had the onset of chestpain. Were it not for this prehospital ECG and the cardiacarrest, the diagnosis may have been significantly delayed. Had this happened, the artery may have re-occluded prior to angiography, with resultant recurrent cardiacarrest and/or shock and death.
He woke up alert and with chestpain which he also had experienced intermittently over the previous few days. The history in today's case with sudden loss of consciousness followed by chestpain is very suggestive of ACS and type I ischemia as the cause of the ECG changes. What do you think?
Subendocardial Ischemia from another Cause ( ie, sustained tachyarrhythmia; cardiacarrest; shock or profound hypotension; GI bleeding; anemia; "sick patient" , etc. ). Having looked for negative U waves in patients with chestpain over a period of decades — I'll emphasize that this is not a common finding.
Jesse McLaren (@ECGcases), of Emergency Medicine Cases Reviewed by Pendell Meyers and Steve Smith An 85yo with a history of hypertension developed chestpain and collapsed, and had bystander CPR. On arrival, GCS was 13 and the patient complained of ongoing chestpain. Vitals were HR 58 BP 167/70 R20 sat 96%.
Some patients have baseline RBBB with LAFB, but in patients with likely ACS, these are associated with severe infarction with cardiacarrest, cardiogenic shock or impending shock. Here are some cases of RBBB with LAFB: What is the Diagnosis in this 70-something with ChestPain?
We record ECGs in triage on every patient with chestpain, and some other indications, and this amounts to 8000 ECGs in triage each year, costing at most $200,000 (8000 x $20.00). Given the dire consequences of missing a STEMI or OMI, including cardiacarrest (see cases below), $5700 is extremely cheap.
Written by Pendell Meyers A man in his 70s with no cardiac history presented with acute weakness, syncope, and fever. He denied chestpain or shortness of breath. In the clinical context of weakness and fever, without chestpain or shortness of breath, the likelihood of Brugada pattern is obviously much higher.
There was no chestpain. Unfortunately, the patient had a cardiacarrest on arrival to the cath lab, before return of the potassium. A patient who does not present with chestpain should be particularly scrutinized for other causes of the ECG abnormalities. The computer read inferior MI.
While many arrhythmias are harmless, some can be life-threatening and increase your risk of stroke, heart failure, and sudden cardiacarrest. This can lead to chestpain (angina) and increase your risk of heart attack or stroke, especially if you already have underlying heart disease.
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