Remove Cardiac Arrest Remove Cardiogenic Shock Remove Ultrasound
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Extreme shock and cardiac arrest in COVID patient

Dr. Smith's ECG Blog

A bedside cardiac ultrasound was normal, with no effusion. Lactate was 20, POC Cardiac US showed EF estimated at 30%, and formal echo showed EF of only 15%, and a normal RV. Assessment was severe sudden cardiogenic shock. Clinically — the patient was felt to be in cardiogenic shock. What is it?

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What are treatment options for this rhythm, when all else fails?

Dr. Smith's ECG Blog

The patient in today’s case presented in cardiogenic shock from proximal LAD occlusion, in conjunction with a subtotally stenosed LMCA. Another approach is sympathetic chain (stellate ganglion) blockade if you have the skills to do it: it requires some expertise and ultrasound guidance. RCA — 100% proximal occlussion.

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LBBB: Using the (Smith) Modified Sgarbossa Criteria would have saved this man's life

Dr. Smith's ECG Blog

But the lack of traditional Sgarbossa criteria is not reassuring enough for such high pretest probability (elderly patient with chest pain, out of hospital cardiac arrest and LBBB), and the Modified Sgarbossa Criteria confirms Occlusion MI in this case. But by this time the patient went into cardiogenic shock and passed away.

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Two patients with chest pain and RBBB: do either have occlusion MI?

Dr. Smith's ECG Blog

I would do bedside ultrasound to look at the RV, look for B lines as a cause of hypoxia (which would support OMI, and argue against PE), and if any doubt persists, a rapid CT pulmonary angiogram. There was 100% proximal LAD occlusion with TIMI 0 flow, and cardiac arrest in the cath lab. There is sinus tachycardia at ~100/minute.

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90 year old with acute chest and epigastric pain, and diffuse ST depression with reciprocal STE in aVR: activate the cath lab?

Dr. Smith's ECG Blog

An elderly man with sudden cardiogenic shock, diffuse ST depressions, and STE in aVR Literature 1. Thirty-six patients (36%) presented with cardiac arrest, and 78% (28/36) underwent emergent angiography. Widespread ST-depression with reciprocal aVR ST-elevation can be cause by: Heart rate related: tachyarrhythmia (e.g.,