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A bedside cardiacultrasound was normal, with no effusion. Lactate was 20, POC Cardiac US showed EF estimated at 30%, and formal echo showed EF of only 15%, and a normal RV. Assessment was severe sudden cardiogenicshock. Clinically — the patient was felt to be in cardiogenicshock. What is it?
The patient in today’s case presented in cardiogenicshock from proximal LAD occlusion, in conjunction with a subtotally stenosed LMCA. Another approach is sympathetic chain (stellate ganglion) blockade if you have the skills to do it: it requires some expertise and ultrasound guidance. RCA — 100% proximal occlussion.
But the lack of traditional Sgarbossa criteria is not reassuring enough for such high pretest probability (elderly patient with chest pain, out of hospital cardiacarrest and LBBB), and the Modified Sgarbossa Criteria confirms Occlusion MI in this case. But by this time the patient went into cardiogenicshock and passed away.
I would do bedside ultrasound to look at the RV, look for B lines as a cause of hypoxia (which would support OMI, and argue against PE), and if any doubt persists, a rapid CT pulmonary angiogram. There was 100% proximal LAD occlusion with TIMI 0 flow, and cardiacarrest in the cath lab. There is sinus tachycardia at ~100/minute.
An elderly man with sudden cardiogenicshock, diffuse ST depressions, and STE in aVR Literature 1. Thirty-six patients (36%) presented with cardiacarrest, and 78% (28/36) underwent emergent angiography. Widespread ST-depression with reciprocal aVR ST-elevation can be cause by: Heart rate related: tachyarrhythmia (e.g.,
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