Cardiac Arrest Cardiogenic Shock Tachycardia 
Dr. Smith's ECG Blog
OCTOBER 20, 2024
Figure B At this point, with the ECG changing from diffuse ST depression to widespread ST elevation and the patient presenting in cardiogenic shock, left main coronary artery (LMCA) occlusion is the likely diagnosis. This patient is actively dying from a left main coronary artery OMI and cardiac arrest from VT/VF or PEA is imminent!
Dr. Smith's ECG Blog
NOVEMBER 12, 2020
Lactate was 20, POC Cardiac US showed EF estimated at 30%, and formal echo showed EF of only 15%, and a normal RV. Assessment was severe sudden cardiogenic shock. Clinically — the patient was felt to be in cardiogenic shock. They recorded an EC G: New ST Elevation. What is it? There is STE in V2-V6.
Dr. Smith's ECG Blog
JULY 21, 2024
The patient in today’s case presented in cardiogenic shock from proximal LAD occlusion, in conjunction with a subtotally stenosed LMCA. This progressed to electrical storm , with incessant PolyMorphic Ventricular Tachycardia ( PMVT ) and recurrent episodes of Ventricular Fibrillation ( VFib ). RCA — 100% proximal occlussion.
Dr. Smith's ECG Blog
NOVEMBER 11, 2022
There is sinus tachycardia (do not be fooled into thinking this is VT or another wide complex tachycardia!) This pattern is essentially always accompanied by cardiogenic shock and high rates of VT/VF arrest, etc. The patient arrived to the ED in cardiogenic shock but awake. Code STEMI was activated.
Dr. Smith's ECG Blog
OCTOBER 8, 2023
The patient died of cardiogenic shock within 24 hours despite mechanical circulatory support. Smith: This bizarre ECG looks like a post cardiac arrest ECG with probable acidosis or hyperkalemia in addition to OMI. This patient at cath had a large CX occlusion with a massive troponin release. Troponin T >42.000ng/L.
Dr. Smith's ECG Blog
OCTOBER 17, 2024
There was 100% proximal LAD occlusion with TIMI 0 flow, and cardiac arrest in the cath lab. There is sinus tachycardia at ~100/minute. As often emphasized by Dr. Smith — sinus tachycardia is not a common finding with acute OMI unless something else is going on (ie, cardiogenic shock ). As per Dr.
EMS 12-Lead
JULY 24, 2023
Category 2 : An increase in myocardial oxygen demand due to tachycardia, elevated ventricular afterload (BP or aortic stenosis), or increased wall stretch (admittedly this latter is more complicated) or a decrease in oxygen supply due to hypotension, anemia, hypoxia, or a combination of all of the above. This results in Type I MI.
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