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There is sinus bradycardia with one PVC. The ways to tell for certain include intravascular ultrasound (to look for extra-luminal plaque with rupture) or "optical coherence tomography," something I am entirely unfamiliar with. If there is any evidence of atherosclerosis, modifiable CAD riskfactors should be treated aggressively.
Bedside ultrasound showed no effusion and moderately decreased LV function, with B-lines of pulmonary edema. There is also bradycardia. Bradycardia puts patients at risk for "pause-dependent" Torsades de Pointes. He was managed medically with Clopidogrel. He appeared to be in shock.
Check : [vitals, SOB, Chest Pain, Ultrasound] If the patient has Abdominal Pain, Chest Pain, Dyspnea or Hypoxemia, Headache, Hypotension , then these should be considered the primary chief complaint (not syncope). Aortic Dissection, Valvular (especially Aortic Stenosis), Tamponade. Frequent or repetitive PACs ii. orthostatic vitals b.
--Genetic testing could be helpful to confirm the diagnosis and allow for screening of other at-risk family members. --EP EP study to further risk stratify her is recommended, with ICD placement depending on the results. Conclusion of this paper: Fever is a great riskfactor for arrhythmia events in Brugada Syndrome patients.
There is also STE in V1 which is diagnostic of right ventricular OMI in this situation , and partly explains the syncope and hypotension (along with the bradycardia). There is a trend toward these patients being younger with a greater relative percentage of women and fewer traditional cardiac riskfactors. Embolism with lysis.
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