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Both of these features make inferior + RV MI by far the most likely ( Pseudoanteroseptal MI is another name for this ) There is also sinus bradycardia and t he patient is in shock with hypotension. A narrow complex bradycardia without any P-waves is also likely to respond to atropine, as it may be a junctional rhythm.
There are multiple possible clinical situations that could account for diffuse subendocardial ischemia that is not due to ACS and plaque rupture. As per Dr. Nossen — today's initial ECG ( LEFT tracing in Figure-2 ) shows sinus bradycardia with QRS widening due to bifascicular block ( RBBB/LAHB ).
A fast heartbeat is called tachycardia, while a slow heartbeat is called bradycardia in medical terms. Coronary artery disease Excessive cholesterol builds up plaque that blocks the arteries supplying blood to the heart. Arrhythmia In simple words, arrhythmia refers to an irregular heartbeat.
During observation in the ED the patient had multiple self-terminating runs of Non-Sustained monomorphic Ventricular Tachycardia (NSVT). Below in Figure-5 is a 10-minute continuous lead II recording on oral Flecainide, now showing sinus bradycardia without a single PVC! There were no plaques or stenoses. No PVCs are seen.
Within ten minutes, she developed bradycardia, hypotension, and ST changes on monitor. Bradycardia and heart block are very common in RCA OMI. Mechanisms of plaque formation and rupture. Coronary plaque disruption. He told the patient this horrible news. The other point in favor of RCA is junctional rhythm. Virmani, R.,
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