Remove Bradycardia Remove Exercise Remove Pacemaker
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What to Know About Bradycardia

AMS Cardiology

This can include our hearts, which may develop conditions like bradycardia or a slow heart rate. What Is Bradycardia ? Are you wondering “ What is bradycardia ?” Bradycardia is a condition in which the heart’s rhythm is too slow. Medications – Certain medications can slow down the heart rate as a side effect.

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ECG Blog #450 — A "Healthy" 30yo with Dizziness

Ken Grauer, MD

To improve visualization — I've digitized the original ECG using PMcardio ) MY Thoughts on the ECG in Figure-1: This is a challenging tracing to interpret — because there is marked bradycardia with an irregular rhythm and a change in QRS morphology. Figure-1: The initial ECG in today's case. ( The QRS complex is wide ( ie, >0.10

Blog 103
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Cardiomatics guide: Analyzing arrhythmias made easy

Cardiomatics

Interpreting the waves and detecting abnormalities: Typically, the heart conducts electricity in a pathway starting in the sinoatrial node (SA), our heart’s “natural pacemaker”, located in the wall of the right atrium. In healthy individuals occurs during exercising or strong emotions. Usually does not exceed 160 bpm.

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What is athlete’s heart?

All About Cardiovascular System and Disorders

During aerobic exercise which is isotonic, the heart rate and stroke volume increases. Isometric exercise or weight training on the other hand causes only slight increase in cardiac output due to increase in heart rate. Effect of exercise on right ventricle. J point elevation and early repolarization pattern has been reported.

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A man in his 40s with epigastric pain and ST Elevation

Dr. Smith's ECG Blog

After the heart rate increased slightly, here was the repeat ECG: Sinus bradycardia, only slightly faster rate than prior. That said — what is unusual about the rhythm in the initial ECG of today's case — is the marked bradycardia! Whether this is the result of a vasovagal reaction to the patient's abdominal pain? —

Blog 52
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Syncope and Block

EMS 12-Lead

He received a permanent pacemaker during the subsequent inpatient stay. plaque disruption), the T waves still manifest markings of a previous state of suboptimal coronary flow that resolved: Type II supply-demand mismatch in the setting of extreme bradycardia. Hospital transport was unremarkable.

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What is strange about this paroxysmal atrial fibrillation in an otherwise healthy patient? And what happened after giving ibutilide?

Dr. Smith's ECG Blog

Whereas at low to moderate degrees of exercise, the risk of developing AFib in younger athletic individuals is reduced — there appears to be a “threshold” for exercise intensity with longterm endurance training, beyond which the risk of developing AFib paradoxically increases!