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CT of the chest showed no pulmonaryembolism but bibasilar infiltrates. Discontinue all negative chronotropic agents, since the risk of torsade is much higher with bradycardia or pauses. She was intubated. Bedside cardiac ultrasound showed moderately decreased LV function. The plan: 1. Place temporary pacemaker 3.
The receiving staff suspects pulmonaryembolism due to S1Q3T3 on the ECG and administers TPA. The patient did have massive pulmonary emboli, but he also had profound intraventricular and subarachnoid hemorrhages. Learning points: TCP is primarily recommended for bradycardia that does not respond to atropine, or other agents.
In any case, there is bradycardia. It makes pulmonaryembolism (PE) very likely. The small LV implies very low LV filling pressures, which implies low pulmonary venous pressure. But it is bradyasystolic, so pulmonaryembolism must be high on the differential. No shock was ever delivered.
The D-dimer was elevated at 942, and the subsequent CT angiogram of the chest showed bilateral lower lobe subsegmental pulmonary emboli with a small right pleural effusion. Normal 0 false false false EN-US X-NONE X-NONE Normal 0 false false false EN-US X-NONE X-NONE The final diagnosis on his ED note: pulmonaryembolism AND pericarditis.
On his physical examination, cardiac and pulmonary auscultation was completely normal. His first electrocardiogram ( ECG) is given below: --Sinus bradycardia. Bi-phasic scan showed no dissection or pulmonaryembolism. Peripheral pulses were all palpable. As he seemed very agitated, fentanyl and diazepam were given.
There is sinus bradycardia with one PVC. pulmonaryembolism, sepsis, etc.), Coronary thrombosis or embolism can result in MINOCA, either with or without a hypercoagulable state. She then had a 12-lead: What do you think? The diagnosis of MINOCA should exclude: 1) other overt causes for elevated troponin (e.g.,
PVCs N ot generally considered abnormal ECG findings: Isolated PAC, First Degree AV Block, Sinus bradycardia at a rate of 35-45, and Nonspecific ST-T abnormalities (even if different from a previous ECG). Thus, if there is documented sinus bradycardia, and no suspicion of high grade AV block, at the time of the syncope, this is very useful.
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