Bradycardia Embolism Ischemia 
Dr. Smith's ECG Blog
JANUARY 2, 2025
CT of the chest showed no pulmonary embolism but bibasilar infiltrates. Discontinue all negative chronotropic agents, since the risk of torsade is much higher with bradycardia or pauses. She was intubated. Bedside cardiac ultrasound showed moderately decreased LV function. The plan: 1. Place temporary pacemaker 3.
Dr. Smith's ECG Blog
DECEMBER 15, 2015
In any case, there is bradycardia. There is ST depression beyond the end of the wide QRS in I, II, aVF, and V4-V6, diagnostic of with subendocardial ischemia. It makes pulmonary embolism (PE) very likely. But it is bradyasystolic, so pulmonary embolism must be high on the differential. There is no ST elevation.
Dr. Smith's ECG Blog
APRIL 8, 2015
Evidence of acute ischemia (may be subtle) vii. PVCs N ot generally considered abnormal ECG findings: Isolated PAC, First Degree AV Block, Sinus bradycardia at a rate of 35-45, and Nonspecific ST-T abnormalities (even if different from a previous ECG). Aortic Dissection, Valvular (especially Aortic Stenosis), Tamponade. Left BBB vi.
Dr. Smith's ECG Blog
APRIL 6, 2025
His rhythm on telemetry seemed to be sinus bradycardia vs junctional rhythm. It did not show pulmonary embolism or intra-abdominal pathology, but it did show this: See the dark area at the bottom of the image? There is Transmural ischemia of Occlusion MI. Here you can also see that there is dense ischemia of the RV.
Dr. Smith's ECG Blog
MARCH 8, 2025
Within ten minutes, she developed bradycardia, hypotension, and ST changes on monitor. Bradycardia and heart block are very common in RCA OMI. Third, a slow motion segment showing delayed, brisk filling of the PDA due to dislodgment of a thrombus from contrast injection and distal embolization.
Expert insights. Personalized for you.
116 
Let's personalize your content