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Notwithstanding many insightful observations, the electrocardiogram (ECG) arguably ignited the big bang in our understanding of cardiac arrhythmias. Using ECG recording and deductive reasoning, our teachers and predecessors classified the bradycardias and tachycardias and proposed many mechanisms, subsequently proven to be correct.
Here, a rare repeated sinus bradycardia event due to escitalopram is first reported. In an 82-year-old female patient with cardiac dysfunction using digoxin, tachycardia (average heart rate of 93 beats/min) was demonstrated by electrocardiogram (ECG). No other drug changes were made during these periods.
Electrocardiogram (ECG) and telemetry revealed junctional bradycardia with heart rate in 30s and sinus pauses (5-7 seconds). He was admitted for further workup of bradycardia. His home medications included metoprolol succinate 25mg daily which was held given bradycardia. He was euvolemic on physical exam.
In a world where technology reigns supreme, one of the most profound tools in medicine remains the irreplaceable electrocardiogram (ECG). Sinus tachycardia – sinus rhythm above 100 bpm is a sinus tachycardia. Sinus bradycardia – sinus rhythm below 60 bpm is a sinus bradycardia.
An electrocardiogram is a machine used to record the heart's electrical activity. A fast heartbeat is called tachycardia, while a slow heartbeat is called bradycardia in medical terms. Electrocardiogram, echocardiogram, and some other tests are done for patients with cardiac arrest. ECG and EKG refer to the same thing.
Otherwise vitals after intubation were only notable for tachycardia. An initial EKG was obtained: Computer read: sinus tachycardia, early acute anterior infarct. Induced Brugada-type electrocardiogram, a sign for imminent malignant arrhythmias. A rectal temperature was obtained which read 107.9 Heart Rhythm, 13(7): 1515-1520. [2]:
There is sinus tachycardia and also a large R-wave in aVR. Drug toxicity , especially diphenhydramine , which has sodium channel blocking effects, and also anticholinergic effects which may result in sinus tachycardia, hyperthermia, delirium, and dry skin. Her temperature was 106 degrees. As part of the workup, she underwent an ECG.
If the patient has Abnormal Vital Signs (fever, hypotension, tachycardia, or tachypnea, or hypoxemia), then these are the primary issue to address, as there is ongoing pathology which must be identified. Thus, if there is documented sinus bradycardia, and no suspicion of high grade AV block, at the time of the syncope, this is very useful.
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