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Bedside cardiac ultrasound showed moderately decreased LV function. Discontinue all negative chronotropic agents, since the risk of torsade is much higher with bradycardia or pauses. She was intubated. CT of the chest showed no pulmonary embolism but bibasilar infiltrates. The plan: 1. Place temporary pacemaker 3.
He required multiple defibrillations within a period of a few hours. This time, the arrhythmia did not spontaneously terminate — but rather degenerated to VFib, requiring defibrillation. Some episodes of PMVT would terminate spontaneously — but on many occasions, the PMVT degenerated to VFib, requiring defibrillation.
Arrival at time 0 ECG 7 min Roomed in hallway at 17 min Moved to room with monitor at 37 min The patient was seen briefly by the physician, who then went to get an ultrasound machine. VF was refractory to amiodarone, lidocaine, double-sequential defibrillation, esmolol, etc. Resuscitative attempts were initiated quickly.
She was unable to be defibrillated but was cannulated and placed on ECMO in our Emergency Department (ECLS - extracorporeal life support). After good ECMO flow was established, she was successfully defibrillated. There is sinus bradycardia with one PVC. An elderly woman had sudden ventricular fibrillation. The K was normal.
Bedside ultrasound showed no effusion and moderately decreased LV function, with B-lines of pulmonary edema. There is also bradycardia. Bradycardia puts patients at risk for "pause-dependent" Torsades de Pointes. Angio had shown some acute disease in the saphenous vein graft to the posterior descending artery off of the RCA.
Triage physician interpretation: -sinus bradycardia -lateral ST depressions While there are lateral ST depressions (V5, V6) the deepest ST depressions are in V4. When the ICD was finally interrogated, the syncopal events and shocks correlated with two VF events that were defibrillated successfully. Triage EKG: What do you think?
Her bedside cardiac ultrasound was normal We decided to cardiovert her since the time of onset was very recent. Baseline bradycardia in endurance athletes limits the use of ß-blockers. But when you see this, you should suspect that the AV node is not well. I signed her out to one of my partners.
Regardless of further evaluation, she should avoid bradycardia, AV nodal blockers, Na channel blockers, and fevers. --If A bedside cardiac ultrasound revealed grossly normal to hyperdynamic systolic function with no obvious areas of wall motion abnormalities.
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