Bradycardia Defibrillator Pulmonary 
Dr. Smith's ECG Blog
JANUARY 2, 2025
CT of the chest showed no pulmonary embolism but bibasilar infiltrates. Discontinue all negative chronotropic agents, since the risk of torsade is much higher with bradycardia or pauses. She was intubated. Bedside cardiac ultrasound showed moderately decreased LV function. The plan: 1. Place temporary pacemaker 3.
Dr. Smith's ECG Blog
MAY 19, 2020
She was unable to be defibrillated but was cannulated and placed on ECMO in our Emergency Department (ECLS - extracorporeal life support). After good ECMO flow was established, she was successfully defibrillated. There is sinus bradycardia with one PVC. pulmonary embolism, sepsis, etc.), The K was normal. myocarditis).
EMS 12-Lead
MARCH 30, 2024
The receiving staff suspects pulmonary embolism due to S1Q3T3 on the ECG and administers TPA. The patient did have massive pulmonary emboli, but he also had profound intraventricular and subarachnoid hemorrhages. Learning points: TCP is primarily recommended for bradycardia that does not respond to atropine, or other agents.
Dr. Smith's ECG Blog
APRIL 8, 2016
Bedside ultrasound showed no effusion and moderately decreased LV function, with B-lines of pulmonary edema. There is also bradycardia. Bradycardia puts patients at risk for "pause-dependent" Torsades de Pointes. Bradycardia puts patients at risk for "pause-dependent" Torsades de Pointes.
Dr. Smith's ECG Blog
FEBRUARY 20, 2025
she had severe pulmonary edema. This is the etiology of the blood from her nose and mouth (frothy bloody pulmonary edema) This is what frothy bloody pulmonary edema looks like. Subarachnoid hemorrhage causes extreme central catecholamine output, resulting in stress cardiomyopathy, just like takotsubo. From this site.
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