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Submitted and written by Alex Bracey, with edits by Pendell Meyers and Steve Smith: I was walking through the criticalcare section of the ED when I overheard a discussion about the following ECG. Here are inferior leads, and aVL, magnified: A closer inspection of the inferior leads and aVL Sinus bradycardia. What do you think?
There is an obvious inferior STEMI, but what else? Besides the obvious inferior STEMI, there is across the precordial leads also, especially in V1. This STE is diagnostic of Right Ventricular STEMI (RV MI). In fact, the STE is widespread, mimicking an anterior STEMI. EKG is pictured below: What do you think?
Here is his ED ECG: There is obvious infero-posterior STEMI. What are you worried about in addition to his STEMI? There is also bradycardia. Bradycardia puts patients at risk for "pause-dependent" Torsades de Pointes. Bradycardia puts patients at risk for "pause-dependent" Torsades de Pointes. Crit Care Med.
He reports that this chest pain feels different than prior chest pain when he had his STEMI/OMI, but is unable to further describe chest pain. Sensitivity was 87% for OMI in our validation study (it was 34% for STEMI criteria). MY Thoughts on the ECG in Figure-1: The rhythm in ECG #1 is sinus bradycardia at ~50-55/minute.
Despite the clinical context, Cardiology was consulted due to concerns for a "STEMI". It is critically important for all EM and criticalcare providers to have an intimate understanding of hyperkalemia and its ECG findings. Peaked T waves: Hyperacute (STEMI) vs. Early Repolarizaton vs. See the diagram above.
Within ten minutes, she developed bradycardia, hypotension, and ST changes on monitor. Bradycardia and heart block are very common in RCA OMI. Multidisciplinary criticalcare management of electrical storm. After this ECG was obtained, the ER physician received word that the patient's husband had died in the crash.
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