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Written by Willy Frick A man in his 50s with history of hypertension, hyperlipidemia, and a 30 pack-year smoking history presented to the ER with 1 hour of acute onset, severe chestpain and diaphoresis. His ECG is shown: What do you think? That is, until the 7th R wave which comes a little bit sooner than expected. What do you think?
Written by Willy Frick A 46 year old man with a history of type 2 diabetes mellitus presented to urgent care with complaint of "chest burning." The documentation does not describe any additional details of the history. The ECG shows sinus bradycardia but is otherwise normal. They also documented "Reproducible chest tenderness."
The combination of absence of chestpain and history of LV aneurysm made it easy to assess that this patient does not have acute OMI. In view of the History with the current admission ( ie, presenting to the ED for vertigo — with no new chestpain ) — I interpreted ECG #1 as no OMI.
60-something with h/o MI and stents presented with chestpain radiating to the back and nausea/vomiting. This is sinus bradycardia. In this patient with documented coronary disease — these q waves could reflect prior lateral infarction ( especially in view of the Q in lead aVL ). Time zero What do you think? Pericarditis?
If a patient presents with chestpain and a normal heart rate, or with shockable cardiac arrest, then ischemic appearing ST elevation is STEMI until proven otherwise. CLICK HERE — for the ESC/ACC/AHA/WHF 2018 Consensus Document on the 4th Universal Definition of MI, in which these concepts are discussed and illustrated.
Written by Colin Jenkins and Nhu-Nguyen Le with edits by Willy Frick and by Smith A 46-year-old male presented to the emergency department with 2 days of heavy substernal chestpain and nausea. He had no previously documented medical problems except polysubstance use. The patient continued having chestpain.
Written by Jesse McLaren A 75 year old with a history of CABG called EMS after 24 hours of chestpain. There’s sinus bradycardia, normal conduction, normal axis, delayed R wave progression, and normal voltages. Sinus bradycardia.” paramedic transportation to the ED as “chestpain, STEMI negative” 2.
The patient in today’s case is a previously healthy 40-something male who contacted EMS due to acute onset crushing chestpain. The pain was 10/10 in intensity radiating bilaterally to the shoulders and also to the left arm and neck. In both tracings — an exceedingly fast PMVT is documented. The below ECG was recorded.
A late middle-aged man presented with one hour of chestpain. There is also bradycardia. Bradycardia puts patients at risk for "pause-dependent" Torsades de Pointes. Torsades in acquired long QT is much more likely in bradycardia because the QT interval following a long pause is longer still.
It was from a patient with chestpain: Note the obvious Brugada pattern. Our patient had a Brugada Type 1 pattern elicited by an elevated core temperature, which is also a documented phenomenon. The elevated troponin was attributed to either type 2 MI or to non-MI acute myocardial injury. There is no further workup at this time.
Check : [vitals, SOB, ChestPain, Ultrasound] If the patient has Abdominal Pain, ChestPain, Dyspnea or Hypoxemia, Headache, Hypotension , then these should be considered the primary chief complaint (not syncope). Aortic Dissection, Valvular (especially Aortic Stenosis), Tamponade. Frequent or repetitive PACs ii.
Thus, Brugada is the likely diagnosis _ A very nice explanation of this is given in the document quoted below on current ECG criteria for Brugada pattern. Regardless of further evaluation, she should avoid bradycardia, AV nodal blockers, Na channel blockers, and fevers. --If Bayes de Luna, A et al.
Case A 68 year old man with a medical history of hypertension, hyperlipidemia, and CAD with stent deployment in the RCA presented to the emergency department with chestpain. A post-cath EKG was recorded at 0719: The computer interpretation read Sinus bradycardia, otherwise normal ECG. He had an EKG recorded right away.
Within ten minutes, she developed bradycardia, hypotension, and ST changes on monitor. Bradycardia and heart block are very common in RCA OMI. He had no chestpain, dyspnea, or any other anginal equivalent, and his vital signs were normal. He told the patient this horrible news. Galiuto, L., Yoshida, T., Manfredini, R.,
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