Bradycardia Cardiomyopathy Ischemia 
Dr. Smith's ECG Blog
JUNE 5, 2024
The ECG does not show any definite signs of ischemia. I thought the complete lack of QTc prolongation and anatomic localization of ECG findings made Takotsubo cardiomyopathy unlikely. It is unclear if the patient was pain free at this time. Initial high sensitivity troponin I returned at 6ng/L (normal 0.20
Dr. Smith's ECG Blog
OCTOBER 18, 2023
During the night, while on telemetry, the patient became bradycardic, with periods of isorhythmic AV dissociation (nodal escape rhythm alternating with sinus bradycardia), and there were sporadic PVCs. Most such rhythms in the setting of ischemia are VF and will not convert without defibrillation. Echo revealed normalized function.
Dr. Smith's ECG Blog
DECEMBER 5, 2016
Description Sinus bradycardia. There is ST elevation in V2 and V3 There are inverted T-waves in V2 and V3 There are prominent U-waves in V2 and V3 Many responders were worried about ischemia or hypertrophic cardiomyopathy. This short QT at least makes ischemia all but impossible. There is high voltage.
Dr. Smith's ECG Blog
JANUARY 2, 2025
And of course Ken's comments at the bottom) An elderly obese woman with cardiomyopathy, Left bundle branch block, and chronic hypercapnea presented hypoxic with altered mental status. I do not see OMI here and all trops were only minimally elevated, consistent with either chronic injury from cardiomyopathy or with acute injury from sepsis.
Dr. Smith's ECG Blog
JUNE 23, 2023
A 50-something male with unspecified history of cardiomyopathy presented in diabetic ketoacidosis (without significant hyperkalemia) with a wide complex tachycardia and hypotension. The fact that he has a cardiomyopathy argues for a more typical ventricular tachycardia, as does the absence of rSR' in lead V1. It is regular.
Dr. Smith's ECG Blog
JANUARY 21, 2023
This ECG shows a sinus bradycardia with a normal conduction pattern (normal PR, normal QRS, and normal QTc), normal axis, normal R-wave progression, normal voltages. Hypothermia can also produce bradycardia and J waves, with a pseudo-STEMI pattern. There is marked sinus bradycardia. What do you think? Pacing Clin Electrophysiol.
EMS 12-Lead
JUNE 1, 2022
Such findings would normally suggest primary ischemia with concomitant surveillance of coronary occlusion, but these ST/T changes might very well be secondary to the Escape mechanism at hand. LBBB is typically the result of preexisting hypertrophy, ischemic heart disease, or cardiomyopathy. 5] Isnard, R. & Pousset, F.
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