Bradycardia Cardiac Arrest Myocardial Infarction 
Dr. Smith's ECG Blog
APRIL 8, 2016
There is also bradycardia. Bradycardia puts patients at risk for "pause-dependent" Torsades de Pointes. Torsades in acquired long QT is much more likely in bradycardia because the QT interval following a long pause is longer still. If cardiac arrest from hypokalemia is imminent (i.e., mEq/L, from 1.9
Dr. Smith's ECG Blog
JULY 21, 2024
Troponin T peaked at 38,398 ng/L ( = a very large myocardial infarction, but not massive-- thanks to the pre-PCI spontaneous reperfusion, and rapid internvention!! ). Some residual ischemia in the infarct border might still be present. Principal adverse cardiac effects of Quinidine include QRS widening and QTc prolongation.
Dr. Smith's ECG Blog
MAY 19, 2020
There is sinus bradycardia with one PVC. There is "Shark Fin morphology" I saw this and thought for certain that this was going to be an LAD or left main occlusion as etiology of arrest, and etiology of profound ST Elevation in I, II, aVL, and V3-V6, and ST depression in III, V1 and V2. She then had a 12-lead: What do you think?
Dr. Smith's ECG Blog
MAY 6, 2019
Only 5-18% of ED patients with chest pain have a myocardial infarction of any kind. Ischemia/infarction. Bradycardia. Could this be a coincidence that the patient was experiencing chest pain and upon ECG capture the atypical brugada pattern was found? Answer : History is insensitive and nonspecific. It helps a little bit.
Dr. Smith's ECG Blog
JANUARY 21, 2023
This ECG shows a sinus bradycardia with a normal conduction pattern (normal PR, normal QRS, and normal QTc), normal axis, normal R-wave progression, normal voltages. Clinical questions : Is this an occlusion myocardial infarction and does the patient need the cath lab? There is marked sinus bradycardia. What do you think?
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