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CASE CONTINUED She was admitted to the ICU. Discontinue all negative chronotropic agents, since the risk of torsade is much higher with bradycardia or pauses. See this post: How a pause can cause cardiacarrest 2. LBBB, ventricular pacing, etc.)." The plan: 1. Place temporary pacemaker 3.
His temperature was brought back to normal over time in the ICU. C), with Cardiac Echo -- A Pathognomonic ECG. -- Read this ECG -- Osborn Waves and Hypothermia (this is the "Figure" above) What does LBBB look like in severe hypothermia? He was extubated and had normal neurologic function. He did well and was discharged.
He was admitted to the ICU and was unstable, in shock, overnight. Case Continued: He was stabilized on more calcium, pressors, and high dose insulin. 3 hours later, this was recorded at a K of 2.8 mEq/L and total calcium of 14.7 mg/dL: The massive ST Elevation persists. What is going on here?
Written by Pendell Meyers, with edits by Steve Smith Thanks to my attending Nic Thompson who superbly led this resuscitation We received a call that a middle aged male in cardiacarrest was 5 minutes out. No other cause of arrest was identified based on lab results or pan-CT scan.
One hour later (labs not yet returned), here is the ECG recorded just after the team noticed a sudden wide complex with precipitous decompensation, just before cardiacarrest: Bizarre, Brady, and Broad (wide QRS). Upon arrival in the ICU, before getting Continuous Veno-Venous Hemodialysis (CVVHD), his potassium had risen again to 7.8
The patient was admitted to the ICU for close monitoring and electrolyte repletion and had an uneventful hospital course. Polymorphic Ventricular Tachycardia Long QT Syndrome with Continuously Recurrent Polymorphic VT: Management CardiacArrest. See these other relevant cases: What are these bizarre bigeminal PVCs?? Is it STEMI?
He was admitted to the ICU and transferred emergently to a facility where he could undergo emergent dialysis as a part of further evaluation and management. Steve, what do you think of this ECG in this CardiacArrest Patient?" HyperKalemia with CardiacArrest. The QRS is narrow and T waves are much less peaked.
Theres sinus bradycardia, borderline PR interval, narrow QRS; normal axis/R wave progression; low precordial voltages, and subtle peaked T waves (most obvious in V2, but all T waves are symmetric with a narrow base). Theres no prior ECG to compare - but the bradycardia, prolonged PR and peaked T waves could all be from hyperkalemia.
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