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He presented to the Emergency Department with a bloodpressure of 111/66 and a pulse of 117. He was rushed by residents into our critical care room with a diagnosis of STEMI, and they handed me this ECG: There is sinus tachycardia with ST elevation in II, III, and aVF, as well as V4-V6. He had this ECG recorded.
A male in his 40's who had been discharged 6 hours prior after stenting of an inferoposterior STEMI had sudden severe SOB at home 2 hours prior to calling 911. Bloodpressure was 215/124 and HR 115 (on metoprolol). Here is his ED ECG: There is sinus tachycardia. Is this acute STEMI? He had no chest pain.
His previous echo one month prior shows the same thing: “consistent with old infarct in LAD vascular territory, with EF 45%” "I think there is something else causing his tachycardia which is exaggerating his EKG findings and mimicking an acute myocardial infarction." The patient spontaneously converted back to sinus tachycardia.
The bloodpressure was 110/60. The "criteria" for posterior STEMI are 0.5 Is it STEMI or NonSTEMI? The patient had no hypertension, no tachycardia, a normal hemoglobin, no drug use, no hypotension/shock, no murmur of aortic stenosis. It was not relieved by anything. He had no previous medical history.
Given that there was such a high bloodpressure, it is possible that this is a type 2 MI (supply demand mismatch due to high oxygen demand when myocardium is pumping against such elevated bloodpressure.) Thus, Wellens' syndrome should be thought of as a transient OMI or transient STEMI. The patient was admitted.
The initial bloodpressure was 80/palp with a heart rate of 104, respirations 20, oxygen saturations of 94% and a finger stick blood glucose of 268. Clinical Course The paramedic activated a “Code STEMI” alert and transported the patient nearly 50 miles to the closest tertiary medical center.
The cardiac monitor showed sinus rhythm but the automatic bloodpressure cuff was not reading. We arrived in the resuscitation bay and recorded a heart rate of 115 bpm and bloodpressure of 50/30 mm Hg. Why would she have such varying bloodpressure? She was discharged home in good condition.
He is placed on heparin drip, he will have IV beta-blocker and oral beta blocker for heart rate control and bloodpressure management. There is sinus tachycardia at 100-105/minute. Admitted to the hospital service for further evaluation and management." Figure-1: Comparison of the first 3 ECGs in today's case.
The ECG shows obvious STEMI(+) OMI due to probable proximal LAD occlusion. Immediately after contrast injection into the LMCA, the patient had circulatory collapse, with a precipitous drop in bloodpressure. An Impella device was placed to maintain cardiac output and perfusion pressures. The below ECG was recorded.
There is an obvious inferior STEMI, but what else? Besides the obvious inferior STEMI, there is across the precordial leads also, especially in V1. This STE is diagnostic of Right Ventricular STEMI (RV MI). In fact, the STE is widespread, mimicking an anterior STEMI. EKG is pictured below: What do you think?
pre-existing, stable atherosclerosis) amidst any state of global duress – to include hypertension, hypoxia, tachycardia, hypotension, sepsis, and GI bleed, for example. Although the bloodpressure resolved, his pain, however, did not. STEMI was activated and the patient went to Cath on arrival.
On arrival in the ED, he was hypotensive with a systolic bloodpressure in the 70s. Despite the clinical context, Cardiology was consulted due to concerns for a "STEMI". After initiating treatment for hyperkalemia, repeat ECG showed resolution of Brugada pattern: The ECG shows sinus tachycardia. What is the Rhythm?
Her bloodpressure on arrival was 153/69. The status of the patients chest pain at this time is unknown : EKG 1, 1300: There is sinus tachycardia and artifact of low and high frequency. However, there is also significant tachycardia , with heart rate of 116, and known hypoxia. Also see these posts of Type II STEMI.
She had this ECG recorded: Obvious massive anterior STEMI She was quickly brought to the critical care area and the cath lab was activated. The bloodpressure was 170/100 in the critical care area. Here is the ECG at 25 minutes: Terrible LAD STEMI (+) OMI So a CT scan was done which of course showed a normal aorta.
His first EKG is shown below, with a lead II rhythm strip: EKG 1, 1645 A provisder who is looking for STEMI would not see much in this EKG. It is possible that the T waves in this EKG are of an intermediate morphology between full-blown STEMI and inferior reperfusion. This is the classic morphology of hyperacute T waves.
PEARL # 3: Knowing there is an acute inferior STEMI I looked next to see if there is also acute posterior involvement ( which so often accompanies inferior MI ). Be ready with prudent administration of IV fluids if bloodpressure drops ( See ECG Blog #190 for more on RV MI, and its different hemodynamics ).
This ECG was recorded: It is difficult to appreciate P-waves, but I believe this is sinus tachycardia. It is correct that he did not have chest pain, but we must remember that fully 1/3 of full blown STEMI do not present with chest pain. This is extremely elevated for a type 2 MI and totally consistent with STEMI.
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