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High bloodpressure, also known as hypertension, is a common condition that affects millions of people worldwide. Often referred to as the silent killer, hypertension can quietly damage your heart and other vital organs over time. What Is High BloodPressure?
When you look at the risk of having a heartattack, it is true that the older you are, the greater the odds of having a heartattack 1. While only 1-2% of those having a heartattack are less than 65 years of age, 1-2% of this very large number means a LOT of heartattacks. So far, so good.
This forces the heart to work harder to supply oxygen to tissues and organs. Increased Heart Rate and BloodPressure: Nicotine stimulates the adrenal glands to release adrenaline, which increases heart rate and bloodpressure. Over time, this constant strain can damage the heart and arteries.
You cannot eliminate the plaque entirely, but multiple clinical trials have shown plaque regression using high-intensity cholesterol-lowering treatments, which I have discussed previously. All of these parameters are important and need to be considered when evaluating plaque regression. REVERSAL Investigators.
Vascular plaque. Perhaps your bloodpressure has been a little too high for a little too long, putting strain on your blood vessels. It starts with inflammation.
Here are some of the major ways in which chronic stress negatively impacts cardiovascular health: High BloodPressure When you experience stress, your body releases hormones like cortisol and adrenaline, which cause your heart rate and bloodpressure to spike.
Maintaining cardiovascular health reduces the risk of developing various heart diseases, including heartattack, stroke, and high bloodpressure. Moreover, a healthy heart contributes to improved overall fitness, endurance, and quality of life.
We all want to be heart-healthy and ensuring our cholesterol levels are in the normal range is one of the most critical steps. High cholesterol can increase your risk of severe conditions like heart disease and heartattacks. Simple protein switches can go a long way in improving your heart health. Exercise more.
This is about estimating your near-term risk of a heartattack. The most accurate way (But not the only way) to answer this question is whether or not you have plaque in your coronary arteries. If you already have plaque, your risk of event an event goes up proportional to the amount of plaque you have 2.
“Cholesterol does not cause heart disease.“ “ “Statins do not prevent heartattacks.” In the middle of this hurricane of noise are people who just want to know what to do so they don’t have a heartattack at a young age. “Statins do not prevent heartattacks.”
When discussing heart health, heartattacks and cardiac arrest are two terms that are often mistaken for one another. Understanding the difference between heartattack and cardiac arrest can help in recognizing symptoms, seeking prompt medical care, and even saving lives. What is a HeartAttack?
BloodPressure High bloodpressure is the risk factor responsible for the greatest number of deaths worldwide 2. For every 20mmHg increase in systolic (Top Number) bloodpressure, the risk of dying from a heartattack or stroke doubles 3. Bloodpressure is easy to check.
Primary prevention is the management of the risk factors, e.g. high bloodpressure, early in life to prevent complications of the condition, i.e. coronary artery disease. This refers to all the steps necessary to reduce the odds of a subsequent event, such as a second heartattack or stroke. Secondary prevention.
. ‘ Snipers Alley ’, it turns out, is an age between 40-60, where mostly males were having fatal heartattacks. These patients were not overly bothered about having a heartattack at age 80, but usually, one of their friends, aged 52 or so, had just had a heartattack, and they did not want to be next.
Understanding the Genetic Connection to Heart Disease Your genetic makeup plays a vital role in shaping your heart health. Genes influence various biological processes, including cholesterol metabolism, bloodpressure regulation, and the strength and structure of your heart and blood vessels.
Over a long enough time frame, pretty much everyone will get heart disease. By the time you get to age 80, you will almost certainly have evidence of plaque in your coronary arteries - you will have heart disease. But remember: Heart disease doesn’t kill people. Heartattacks do. Normal BloodPressure.
During each consultation with a patient, I would have to explain certain topics related to heart health, and I found myself repeating them over and over. What should my bloodpressure be? What is heart disease? The average age of these heart donors? Heart Disease Doesn’t Kill People. 33 years of age.
To prevent heart disease, you need to know what causes it, how to measure the relevant factors and what to do about them. When we say heart disease, what we really mean is plaque in the artery wall. No heartattacks. That all depends on your overall risk of a future cardiovascular event like a heartattack.
Some groups will state that any heart events at less than 55 years of age for males and less than 65 for females define early heart disease. A heartattack in a 56-year-old male is early in anyone's books. However, the above age cut-offs give a good idea of what we consider the early presentation of heart disease.
Heart disease does not kill people. Heartattacks do. Appreciating this distinction is critical to understanding heart disease. Heart disease is the presence of plaque or atherosclerosis in the coronary arteries. In this instance, a heartattack. But does this approach work?
Everyone starts with no plaque in the coronary arteries, but over a long enough time frame, everyone develops plaque in their coronary arteries. By age 80, almost everyone will have evidence of advanced plaque in their coronary arteries, as defined by a cardiac CT 1. Plaque accumulation happens in stages. You got it.
Atherosclerotic cardiovascular disease (ASCVD), caused by plaque buildup in arterial walls, is one of the leading causes of disability and death worldwide.1,2 3 Patients with ASCVD are at a higher risk for major adverse cardiovascular events (MACE) including heartattack or myocardial infarction (MI), stroke, and cardiovascular (CV) death.4
A new joint guideline from the American Heart Association (AHA), the American College of Cardiology (ACC) and nine other medical societies reports early diagnosis and treatment of peripheral artery disease is essential to improve outcomes and reduce amputation risk, heartattack, stroke and death for people with Peripheral Artery Disease (PAD).
CT coronary angiography, in addition to a CT CAC, is arguably the best test for estimating whether someone has evidence of coronary artery disease and what that means for their near-term risk of a heartattack. Mixed Plaque - A combination of both calcified and NON-calcified plaque. More often than you would think!
people from the general population), coronary artery calcium scores (CACS) are higher, indicating more calcification and the presence of atherosclerotic plaques. Calcified plaques are known to be more stable and less prone to rupture and lead to a heartattack. When comparing athletes to control groups (i.e.,
If the pump is unable to pump blood out (either because it is defective or because something is making it more difficult to pump blood out in some way) then less blood goes around and this can damage our vital organs and be dangerous. The plaques can damage us in 2 ways.
A higher cumulative LDL cholesterol exposure equals a higher likelihood of plaque in the coronary arteries, known as atherosclerosis. But remember, heart disease or atherosclerosis does not kill people. Heartattacks kill people. But the more plaque you have, the higher the risk of a heartattack.
The mistake most people make when it comes to heart disease is thinking that when someone has a heartattack that, the condition of ‘heart disease’ just appeared. Heartattacks present suddenly. But heart disease presents slowly. The more plaque, the higher the risk.
In the study, post-menopausal women underwent heart scans to assess their CAC score, a measure of plaque buildup—fat, calcium and other substances—in the heart’s arteries. A higher CAC score indicates a higher risk of a heartattack or other cardiac events.
Cross-sectional studies reveal that endurance athletes, particularly middle-aged and older men, often exhibit higher coronary artery calcium scores (CACS) and plaque prevalence compared to less-active individuals. Notably, athletes engaging in very vigorous-intensity exercise are more likely to develop calcified plaques.
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