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Introduction Multiple abnormal electrocardiographic findings have been documented in patients experiencing acute pulmonary embolism. Although sinus tachycardia is the most commonly encountered rhythmic disturbance, subsequent reports have highlighted other findings. Echocardiography confirmed signs of right ventricular dysfunction.
Notice I did not say "pulmonary embolism," because any form of severe acute right heart strain may produce this ECG. Differences of Pulmonary Embolism T-waves from Wellens' T-waves: 1. Acute coronary occlusion (especially during reperfusion) is very rarely accompanied by tachycardia. What is the answer?
The bloodpressure was 110/60. The patient had no hypertension, no tachycardia, a normal hemoglobin, no drug use, no hypotension/shock, no murmur of aortic stenosis. It was not relieved by anything. The pain was not positional, pleuritic, or reproducible. He had no previous medical history. Is it STEMI or NonSTEMI?
The bedside echo showed a large RV (Does this mean there is a pulmonary embolism as the etiology?) When you suspect pulmonary embolism due to large RV on POCUS, always look for right axis deviation and a large R-wave in V1 because the large RV may be entirely due to chronic RVH, not acute PE. 2) Norepinephine to support BloodPressure.
Given that there was such a high bloodpressure, it is possible that this is a type 2 MI (supply demand mismatch due to high oxygen demand when myocardium is pumping against such elevated bloodpressure.) Her initial cTnI returned at 0.25 Wellens' syndrome will almost always develop elevated troponins.
If the patient has Abnormal Vital Signs (fever, hypotension, tachycardia, or tachypnea, or hypoxemia), then these are the primary issue to address, as there is ongoing pathology which must be identified. Any ED systolic bloodpressure less than 90 or greater than 180 mm Hg (+1) 4. h/o heart disease (+1) 3.
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