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He presented to the Emergency Department with a bloodpressure of 111/66 and a pulse of 117. He was rushed by residents into our critical care room with a diagnosis of STEMI, and they handed me this ECG: There is sinus tachycardia with ST elevation in II, III, and aVF, as well as V4-V6. He had this ECG recorded. The HCO3 was 8.
MY Thoughts on this CASE: Not being there — I am unaware of physical exam parameters ( bloodpressure, respiratory rate; oxygen saturation; heart and lung auscultation, etc. ). I see the following: There is sinus tachycardia ( upright P wave with fixed PR interval in lead II ) — at the rapid rate of ~130/minute.
Because of the tachcardia, I would expect her to be very poor left ventricular function and maybe Cardiogenicshock. Still Irregular Bloodpressure during these rhythms was adequate; there was no shock. Still Irregular Bloodpressure during these rhythms was adequate; there was no shock.
The initial bloodpressure was 80/palp with a heart rate of 104, respirations 20, oxygen saturations of 94% and a finger stick blood glucose of 268. In addition, the patient received 750 mL of fluid resuscitation with transient improvement of bloodpressure. If you can use Doppler, then you can diagnose it.
The findings include sinus tachycardia, characteristic QRS morphology most diagnostic in V3 with a small R wave followed by a very large S wave with a convex upward ST segment morphology, ST segment strain morphology in the inferior and anterior leads leading to deep symmetric T-wave inversion. and tachycardia, 1.8. incomplete RBBB 1.7
Figure B At this point, with the ECG changing from diffuse ST depression to widespread ST elevation and the patient presenting in cardiogenicshock, left main coronary artery (LMCA) occlusion is the likely diagnosis. The arterial blood gas showed a lactic acidosis with a lactate level of 17mmol/L. This is an ominous sign.
Immediately after contrast injection into the LMCA, the patient had circulatory collapse, with a precipitous drop in bloodpressure. An Impella device was placed to maintain cardiac output and perfusion pressures. You can see Left Main and Proximal LAD obstruction, but with some flow, which is saving this patient's life.
Why is the patient in shock? He was in profound cardiogenicshock. Both of these features make inferior + RV MI by far the most likely ( Pseudoanteroseptal MI is another name for this ) There is also sinus bradycardia and t he patient is in shock with hypotension. There is an obvious inferior STEMI, but what else?
However, recent studies have observed that people below 40 are also experiencing heart attacks due to high bloodpressure, cholesterol, diabetes, smoking, obesity, a sedentary lifestyle, and social and mental stress. This indicates that restoring normal blood circulation as quickly as possible will result in less damage.
Her bloodpressure on arrival was 153/69. The status of the patients chest pain at this time is unknown : EKG 1, 1300: There is sinus tachycardia and artifact of low and high frequency. However, there is also significant tachycardia , with heart rate of 116, and known hypoxia. An EKG was immediately recorded.
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