Remove Blood Pressure Remove Bradycardia Remove Cardiac Arrest
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Transcutaneous Pacing: Part 2

EMS 12-Lead

Patient had an unwitnessed cardiac arrest without bystander CPR performed. Crew notifies the received ED of an incoming post-arrest patient and notes a sinus bradycardia on their monitor, as seen in Figure 2. Figure 2 : This rhythm shows a sinus bradycardia at a rate between 30 and 40bpm.

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A middle aged man with unwitnessed cardiac arrest

Dr. Smith's ECG Blog

Written by Pendell Meyers, with edits by Steve Smith Thanks to my attending Nic Thompson who superbly led this resuscitation We received a call that a middle aged male in cardiac arrest was 5 minutes out. He was estimated to be in his 50s, with no known PMHx. He arrived with chest compressions ongoing, intubated, and being bagged.

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Could you have prevented this young man's cardiac arrest?

Dr. Smith's ECG Blog

One hour later (labs not yet returned), here is the ECG recorded just after the team noticed a sudden wide complex with precipitous decompensation, just before cardiac arrest: Bizarre, Brady, and Broad (wide QRS). Compartment pressures in the right calf were all 40-50 mmHg. Unfortunately, this was not recognized at this time.

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See what happens when a left main thrombus evolves from subtotal occlusion to total occlusion.

Dr. Smith's ECG Blog

On arrival in the emergency department, invasive blood pressure was 35/15mmHg and the patient was in profound cardiogenic shock with severe confusion secondary to brain hypoperfusion. The arterial blood gas showed a lactic acidosis with a lactate level of 17mmol/L. PUSH THE LYTICS ! The below ECG (ECG #4) was recorded.

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What are treatment options for this rhythm, when all else fails?

Dr. Smith's ECG Blog

Immediately after contrast injection into the LMCA, the patient had circulatory collapse, with a precipitous drop in blood pressure. An Impella device was placed to maintain cardiac output and perfusion pressures. There was no evidence bradycardia leading up to the runs of PMVT ( as tends to occur with Torsades ).

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Another deadly and confusing ECG. Are you still one of the many people who will be fooled by this ECG, or do you recognize it instantly?

Dr. Smith's ECG Blog

On arrival in the ED, he was hypotensive with a systolic blood pressure in the 70s. Hyperkalemia causes peaked T waves and the "killer B's of hyperkalemia", including bradycardia, broad QRS complexes, blocks of the AV node and bundle branches, Brugada morphology, and otherwise bizarre morphology including sine wave.

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Unresponsive and Acidotic: OMI? Acute, subacute, or reperfused? What is the rhythm? Why RV dysfunction? Can CT scan help?

Dr. Smith's ECG Blog

They were unable to obtain a blood pressure. His heart rate was in the low 20s and we were also unable to obtain a blood pressure. He was given 50 mcg epinephrine with good response in both heart rate and blood pressure. His rhythm on telemetry seemed to be sinus bradycardia vs junctional rhythm.