article thumbnail

ECG Blog #415 — The Cath showed NO Occlusion!

Ken Grauer, MD

As discussed in detail in ECG Blog #228 — this seemingly qualifies as a “ Silent ” MI ( Approximately half of those MIs not accompanied by CP — have some other associated symptom such as syncope, which substitutes as a “chest pain equivalent” ). Smith's ECG Blog ). ECG Blog #218 — Reviews HOW to define a T wave as being H yperacute ?

Blog 163
article thumbnail

No Plaque, No Problem: Tackling Atherosclerosis Prevention

Cardiometabolic Health Congress

Stay tuned for our next blog, where we’ll outline actionable solutions to make prevention more effective and accessible. Enhancing Adherence: Simplify treatment plans and build trust through patient education. Driving Change in Clinical Practice: Support providers in adopting evidence-based interventions.

article thumbnail

Dynamic OMI ECG. Negative trops and negative angiogram does not rule out coronary ischemia or ACS.

Dr. Smith's ECG Blog

Thus, it has recently become generally accepted that most plaque ruptures resulting in myocardial infarction occur in plaques that narrow the lumen diameter by 40% of the arterial cross section may be involved by plaque. The pathologist may see a plaque that constitutes, for example, 50% of the cross-sectional area.

Ischemia 123
article thumbnail

Concerning EKG with a Non-obstructive angiogram. What happened?

Dr. Smith's ECG Blog

The commonest causes of MINOCA include: atherosclerotic causes such as plaque rupture or erosion with spontaneous thrombolysis, and non-atherosclerotic causes such as coronary vasospasm (sometimes called variant angina or Prinzmetal's angina), coronary embolism or thrombosis, possibly microvascular dysfunction. It is not rare.

Plaque 128
article thumbnail

The Role of Genetics in Heart Disease: Can You Prevent It?

MIBHS

This blog explores how genetics influence heart health and whether mitigating these inherited risks is possible. Specific genetic variants, such as those affecting cholesterol metabolism, can increase the likelihood of plaque buildup in the arteries.

article thumbnail

Three normal high sensitivity troponins over 4 hours with a "normal ECG"

Dr. Smith's ECG Blog

Although it is statistically unlikely, multiple plaque ruptures are possible. On intravascular ultrasound (IVUS), the mid RCA plaque was described as "cratered, inflamed, and bulky," and the OM plaque was described as "bulky with evidence of inflammation and probably ulceration." Heitner et al. DOI:10.1161/CIRCINTERVENTIONS.118.007305),

Angina 122
article thumbnail

Wide Complex Tachycardia

EMS 12-Lead

I interpreted the ECG as VT with two primary etiological possibilities: 1. Abrupt plaque ulceration of Type 1 ACS leading to VT. Readers of the Smith ECG Blog will probably recognize this a very subtle inferior OMI. The VT vs SVT with Aberrancy debate is beyond the scope of this particular blog post.