ECG Cases 48 – ECG Interpretation in Cardiac Arrest
ECG Cases
FEBRUARY 13, 2024
The post ECG Cases 48 – ECG Interpretation in Cardiac Arrest appeared first on Emergency Medicine Cases.
Blog Cardiac Arrest STEMI 
ECG Blog #415 — The Cath showed NO Occlusion!
Ken Grauer, MD
FEBRUARY 3, 2024
Shortly after arrival in the ED ( E mergency D epartment ) — she suffered a cardiac arrest. BUT — Cardiac catheterization done a little later did not reveal any significant stenosis. Figure-1: The initial ECG in today's case — obtained after successful resuscitation from cardiac arrest. ( No CP ( C hest P ain ).
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ECG Blog #434 — WHY Did this Patient Arrest?
Ken Grauer, MD
JUNE 14, 2024
The ECG in Figure-1 — was obtained from a middle-aged man who presented to the ED ( E mergency D epartment ) in cardiac arrest. Prompt cath is therefore advised if the post-ROSC shows an acute STEMI. To Emphasize: The phenomenon of T-QRS-D is not needed in today's case to recognize the acute STEMI.
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ECG Blog #400 — Is this a NSTEMI?
Ken Grauer, MD
OCTOBER 21, 2023
PEARL # 1: Although seeing an elevated Troponin would provide additional support for immediate cardiac catheterization — the clinical reality is that the initial Troponin reading will not always be elevated in patients with acute coronary occlusion ( See March 24, 2023 post in Dr. Smith's ECG Blog ).
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A man in his 50s with unwitnessed VF arrest, defibrillated to ROSC, and no STEMI criteria on post ROSC ECG. Should he get emergent angiogram?
Dr. Smith's ECG Blog
JULY 9, 2024
Written by Pendell Meyers A man in his 50s was found by his family in cardiac arrest of unknown duration. Despite anticipation by many that the initial post-resuscitation ECG will show an obvious acute infarction — this expected "STEMI picture" is often not seen. Restoration of sinus rhythm is evident in Figure-1.
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Resuscitated from ventricular fibrillation. Should the cath lab be activated?
Dr. Smith's ECG Blog
JULY 25, 2024
But cardiac arrest is a period of near zero flow in the coronary arteries and causes SEVERE ischemia. After cardiac arrest, I ALWAYS wait 15 minutes after an ECG like this and record another. See these related cases: Cardiac arrest, defibrillated, diffuse ST depression and ST Elevation in aVR.
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ECG Blog #381 — Why was the Troponin Normal?
Ken Grauer, MD
MAY 26, 2023
At some point ~1-2 hours after the initial ECG — the patient developed runs of VT, leading to cardiac arrest. ECG Blog #185 — Review of the P s, Q s, 3 R Approach for systematic rhythm interpretation. ECG Blog #271 — Reviews the concept of diffuse Subendocardial Ischemia. ECG Blog #316 — The patient died.
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Guidelines would (erroneously) say that this patient who was defibrillated and resuscitated does not need emergent angiography
Dr. Smith's ECG Blog
JUNE 27, 2024
A patient had a cardiac arrest with ventricular fibrillation and was successfully defibrillated. COACT: The COACT trial was fatally flawed, and because of it, many cardiologists are convinced that if there are no STEMI criteria, the patient does not need to go to the cath lab. These studies did not address OMI ECG findings!!!
Unconscious + STEMI criteria: activate the cath lab?
Dr. Smith's ECG Blog
JANUARY 21, 2023
ECG met STEMI criteria and was labeled STEMI by computer interpretation. J waves can also be induced by Occlusion MI (5), STEMI mimics including takotsubo and myocarditis complicated by ventricular arrhythmias (6, 7), and subarachnoid hemorrhage with VF (8). Take home : Not all STEs are STEMIs or OMIs. What do you think?
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How will you save this critically ill patient? A fundamental and lifesaving ECG interpretation that everyone must recognize instantly.
Dr. Smith's ECG Blog
FEBRUARY 22, 2024
A woman in her 50s with dyspnea and bradycardia A patient with cardiac arrest, ROSC, and right bundle branch block (RBBB). HyperKalemia with Cardiac Arrest. Peaked T waves: Hyperacute (STEMI) vs. Early Repolarizaton vs. Hyperkalemia What will you do for this altered and bradycardic patient?
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Extreme shock and cardiac arrest in COVID patient
Dr. Smith's ECG Blog
NOVEMBER 12, 2020
In a series of 18 patients with COVID and ST elevation, 8 were diagnosed with STEMI, 6 of whom had an angiogram and it showed obstructive coronary disease. 12 All STEMI patients had very high cTn typical of STEMI (cTnT > 1.0 POINT #2: Overall QRS amplitude ( voltage ) is dramatically reduced in ECG #1.
Cardiac arrest, LBBB with STEMI on the ECG, but no Acute Coronary Syndrome!
Dr. Smith's ECG Blog
SEPTEMBER 20, 2011
This 80 year old with a history of CABG had a cardiac arrest. This is as clear a STEMI as you can get. Now, it is true that shortly after a non-ACS cardiac arrest, there can be transient diffuse ST depression, but not ST elevation in a coronary distribution, and there should not be a wall motion abnormality.
A 40-something presented after attempted prehospital resuscitation with persistent Ventricular Fibrillation
Dr. Smith's ECG Blog
JULY 14, 2023
Two recent interventions have proven in randomized trials to improve neurologic survival in cardiac arrest: 1) the combination of the ResQPod and the ResQPump (suction device for compression-decompression CPR -- Lancet 2011 ) and 2) Dual Sequential defibrillation. First — Some thoughts on the post -resuscitation ECG.
A man in his 50s with acute chest pain who is lucky to still be alive.
Dr. Smith's ECG Blog
JUNE 21, 2023
You can subscribe for news and early access (via participating in our studies) to the Queen of Hearts here: [link] queen-form This EMS ECG was transmitted to the nearby Emergency Department where it was remotely reviewed by a physician, who interpreted it as normal, or at least without any features of ischemia or STEMI.
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Saddleback ST Elevation. Is it STEMI? Is it type II Brugada?
Dr. Smith's ECG Blog
NOVEMBER 28, 2018
Saddleback ST Elevation is almost never STEMI 2. An inverted P-wave in lead V2 implies lead misplacement too high Saddleback in STEMI: Here are the only 2 ECGs with V2 "saddleback" that I have ever seen which really represented an LAD Occlusion: Anatomy of a Missed LAD Occlusion (classified as a NonSTEMI) A Very Subtle LAD Occlusion.T-wave
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Missed myocardial infarction with subsequent cardiac arrest
Dr. Smith's ECG Blog
OCTOBER 24, 2013
Appreciation of these subtle ECG findings could have helped to avoid a cardiac arrest and its resulting permanent disability 3. Ischemia on the ECG can be very subtle and is easily missed. Accurate interpretation requires a lot of skill, practice, and experience.
A Patient with Cocaine Chest Pain and Prehospital Computer interpretation of STEMI
Dr. Smith's ECG Blog
JANUARY 25, 2017
911 was called and the medics recorded this ECG (unfortunately, leads V4-V6 are missing) Due to marked ST Elevation, the computer read was STEMI What do you think? See this post and associated case reports: Cardiac arrest, severe acidosis, and a bizarre ECG The patient was admitted and ruled out for acute MI by serial troponins.
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75 year old dialysis patient with nausea, vomiting and lightheadedness
Dr. Smith's ECG Blog
AUGUST 19, 2023
This is diagnostic of infero-posterior OMI, but it is falsely negative by STEMI criteria and with falsely negative posterior leads (though they do show mild ST elevation in V4R). They were less likely to have STEMI on ECG, and more likely to be initially diagnosed as non-ACS.
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What are treatment options for this rhythm, when all else fails?
Dr. Smith's ECG Blog
JULY 21, 2024
The ECG shows obvious STEMI(+) OMI due to probable proximal LAD occlusion. As we've often emphasized on Dr. Smith's ECG Blog — it is rare in practice to see LMCA occlusion, because most such patients die before reaching the hospital. The below ECG was recorded. Figure-1: I've labeled the initial ECG in today's case. (
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90 year old with acute chest and epigastric pain, and diffuse ST depression with reciprocal STE in aVR: activate the cath lab?
Dr. Smith's ECG Blog
NOVEMBER 4, 2022
This has been termed a “STEMI equivalent” and included in STEMI guidelines, suggesting this patient should receive dual anti-platelets, heparin and immediate cath lab activation–or thrombolysis in centres where cath lab is not available. aVR ST segment elevation: acute STEMI or not? aVR ST Segment Elevation: Acute STEMI or Not?
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ECG Blog #383 — Is this Coronary Disease?
Ken Grauer, MD
JUNE 6, 2023
== P lease N OTE : After today — No new ECG Blog posts for 2- to -3 weeks. — I will also not be prompt in replying to emails. == All material on this ECG Blog site remains open! IF you scroll down a little on the right-hand column of this blog — You'll see a lot of icons. Figure-1: The initial ECG in today's case.
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Proportionality is a major element in the ECG Diagnosis of OMI.
Dr. Smith's ECG Blog
DECEMBER 13, 2023
For technical reasons — P waves are not well visible in this tracing — BUT — the P wave in lead I appears to be larger than the P wave in lead II , which is often a tip-off to LA-LL Reversal ( See My Comment in the November 19, 2020 and the May 24, 2022 posts in Dr. Smith's ECG Blog ).
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Cardiac Arrest, acute ST elevation and depression superimposed on LVH, but NOT due to ACS
Dr. Smith's ECG Blog
OCTOBER 4, 2010
Thus, this patient had increased ST elevation (current of injury) superimposed on the ST elevation of LVH and simulating STEMI. The stress of the triathlon cause demand ischemia and ventricular fibrillation. He awoke and did well.
Chest Pain and Right Bundle Branch Block
Dr. Smith's ECG Blog
NOVEMBER 19, 2014
Here are three more dramatic cases that illustrate RBBB + LAFB Case 1 of cardiac arrest with unrecognized STEMI, died. Furthermore, among 35 patients with acute left main coronary artery occlusion, 9 presented with RBBB (mostly with LAH) on the admission ECG.
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What are these hyperacute T waves, with STE and T-wave inversion in aVL, and STD in inferior leads?
Dr. Smith's ECG Blog
FEBRUARY 27, 2023
See many examples of Pseudo STEMI due to hyperkalemia at these two posts: Acute respiratory distress: Correct interpretation of the initial and serial ECG findings, with aggressive management, might have saved his life. We've presented numerous examples of hyperkalemia on Dr. Smith's ECG Blog. No followup EKG was recorded!!
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1 hour of CPR, then ECMO circulation, then successful defibrillation.
Dr. Smith's ECG Blog
MAY 19, 2020
This is a troponin I level that is almost exclusively seen in STEMI. I suspect this is Type 2 MI due to prolonged severe hypotension from cardiac arrest. So this is either a case of MINOCA, or a case of Type II STEMI. If the arrest was caused by acute MI due to plaque rupture, then the diagnosis is MINOCA.
LBBB: Using the (Smith) Modified Sgarbossa Criteria would have saved this man's life
Dr. Smith's ECG Blog
JUNE 11, 2021
The patient was brought to the ED as a possible Code STEMI and was seen directly by cardiology. Similarly, STEMI guidelines call for urgent angiography for refractory ischemia or electrical/hemodynamic instability, regardless of ECG findings. On arrival, GCS was 13 and the patient complained of ongoing chest pain.
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Computer and transferring physician say "normal." What do you think?
Dr. Smith's ECG Blog
AUGUST 1, 2020
He has done a lot of great work on cardiac arrest, including as co-author of our study on esmolol in refractory cardiac arrest, and much more with Keith Lurie. This was sent by Guillaume Debaty, from the Grenoble Region of France. See his Google Scholar profile here. Several of these are present in this case.
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ECG with Aslanger's Pattern. CT Pulmonary Angiogram Reveals LAD Ischemia (Septal Transmural). But this is not Contradictory.
Dr. Smith's ECG Blog
JANUARY 4, 2021
Could this be Septal STEMI (STE in V1 and aVR, with reciprocal ST depression in V4-V6?), In Septal STEMI , transmural ischemia of the septum is recorded by the overlying lead V1 as ST Elevation. Lead III is also on the right and might manifest ST Elevation in Septal STEMI. with ADDED STE in III?
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A Tragic Case, related to the last post
Dr. Smith's ECG Blog
JUNE 15, 2013
The cath lab was activated for STEMI. Unfortunately, the patient had a cardiac arrest on arrival to the cath lab, before return of the potassium. Hyperkalemia alone can cause inferior-posterior pseudo-STEMI: Notice that in both cases, the ST elevation is downsloping and the T-wave is inverted. The potassium returned at 8.4
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Syncope, Shock, AV block, Large RV, "Anterior" ST Elevation.
Dr. Smith's ECG Blog
DECEMBER 15, 2015
It was a PEA or bradyasystolic arrest , not a shockable rhythm. Although most cardiac arrest from MI is due to ventricular fibrillation, some is due to high grade AV block, and so this could indeed be due to large acute STEMI. LV anterior STEMI does not give maximal ST elevation in V1. Is this an anterior (LV) MI?
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PseudoSTEMI and True ST elevation in Right Bundle Branch Block (RBBB). Don't miss case 4 at the bottom.
Dr. Smith's ECG Blog
SEPTEMBER 16, 2015
It may be difficult to read STEMI in the setting of RBBB. There is, however, a long QT also, with abnormal T-waves, but this is not STEMI. This ECG was recorded prehospital, and the computer read STEMI, so the medics activated the cath lab: What do you think? The ECG is consistent with high lateral STEMI. Called 911.
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Chest pain relieved by Maalox and viscous lidocaine
Dr. Smith's ECG Blog
DECEMBER 23, 2014
The patient was diagnosed with esophageal reflux and was being discharged by the nurse when he had a cardiac arrest. Anterolateral STEMI. The formula results in 23.43, just above the 23.4 He was defibrillated. Here is his post resuscitation ECG: Now the diagnosis is obvious.
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Severe shock, obtunded, and a diagnostic prehospital ECG. Also: How did this happen?
Dr. Smith's ECG Blog
NOVEMBER 30, 2019
If cardiac arrest from hypokalemia is imminent (i.e., to greatly decrease risk (although in STEMI, the optimal level is about 4.0-4.5 As I indicated above, in our cardiac arrest case, after pushing 40 mEq, the K only went up to 4.2 Give an initial infusion of 10 mEq IV over 5 minutes; repeat once if needed.
An elderly woman with acute vomiting, presyncope, and hypotension, and a wide QRS complex
Dr. Smith's ECG Blog
NOVEMBER 11, 2022
This pattern is essentially always accompanied by cardiogenic shock and high rates of VT/VF arrest, etc. Code STEMI was activated. Plus recommendations from a 5-member panel on cardiac arrest. A man in his 80s with chest pain What, besides large anterior STEMI, is so ominous about this ECG?
A Middle-Aged male with Chest Pain and an Unusual ECG
Dr. Smith's ECG Blog
MAY 6, 2019
If it is STEMI, it would have to be RBBB with STEMI. This ECG pattern may be diagnostic of B rugada S yndrome IF seen in association with: i ) a history of cardiac arrest; polymorphic VT; or of non-vagal syncope; and / or ii ) a positive family history of sudden death at an early age; and / or iii ) a similar ECG in relatives.
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A man in his 30s with cardiac arrest and STE on the post-ROSC ECG
Dr. Smith's ECG Blog
OCTOBER 10, 2022
He had multiple cardiac arrests with ROSC regained each time. Then there is loss of pulses with continued narrow complex on the monitor ("PEA arrest") Learning Points: Sudden witnessed Cardiac Arrest due to ACS is almost always due to dysrhythmia. This patient arrested shortly after hospital arrival.
Interventionalist at the Receiving Hospital: "No STEMI, no cath. I do not accept the transfer."
Dr. Smith's ECG Blog
SEPTEMBER 21, 2020
Are Some Cardiologists Really Limited by Strict Adherence to STEMI millimeter criteria? I was texted these ECGs by a recent residency graduate after they had all been recorded, along with the following clinical information: A 50-something with no cardiac history, but with h/o Diabetes, was doing physical work when he collapsed.
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Right Bundle Branch Block with Acute ST Elevation Seen Best on Prehospital ECG
Dr. Smith's ECG Blog
FEBRUARY 25, 2009
RBBB in acute STEMI has a very high mortality. Were it not for this prehospital ECG and the cardiac arrest, the diagnosis may have been significantly delayed. Had this happened, the artery may have re-occluded prior to angiography, with resultant recurrent cardiac arrest and/or shock and death.
Tachycardia, fever to 105, and ischemic ST Elevation -- a Bridge too Far
Dr. Smith's ECG Blog
APRIL 15, 2019
A prehospital ECG was recorded (not shown and not seen by me) which was worrisome for STEMI. A previous ECG from 4 years prior was normal: This looks like an anterior STEMI, but it is complicated by tachycardia (which can greatly elevate ST segments) and by the presentation which is of fever and sepsis.
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40-something with severe CP. True + vs. False + high lateral MI. ST depression does not localize.
Dr. Smith's ECG Blog
SEPTEMBER 4, 2016
He had a previous MI with cardiac arrest 2 years prior. Important Learning Point: "STEMI" is defined by millimeter criteria (1 mm in limb leads), which this does not meet. Therefore it is not a STEMI. The ST depression may be the most visibly obvious sign of STEMI. Some are STEMI-equivalents.
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Inferior ST Elevation: what is the Diagnosis?
Dr. Smith's ECG Blog
OCTOBER 5, 2011
If there are no changes in aVL, it is highly unlikely to be inferior STEMI. Would you be certain that it is not STEMI? No one is certain what to do with this information, and it certainly does not impact emergency medicine, in which the problem remains: is it STEMI or not? How would you be able to know this from the ECG alone?
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Cardiologist declines taking patient to the cath lab. Patient dies.
Dr. Smith's ECG Blog
OCTOBER 11, 2020
The provider contacted cardiology to discuss the case, but cardiology "didn't think it was a STEMI, didn't think he needed emergent cath." About two hours after admission, he suffered a cardiac arrest (whether it was VF/VT or PEA is not available) and expired. The whole paradigm is literally called "STEMI" vs. "NSTEMI."
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A man in his 60s with syncope and ST depression. What does the ECG mean?
Dr. Smith's ECG Blog
OCTOBER 25, 2023
Polymorphic Ventricular Tachycardia Long QT Syndrome with Continuously Recurrent Polymorphic VT: Management Cardiac Arrest. A New Seizure in a Healthy 20-something More cases of long QT not measured correctly by computer (these are all fascinating ECGs/cases): Bupropion Overdose Followed by Cardiac Arrest and, Later, ST Elevation.
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