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IMPRESSION: The finding of sinus bradycardia with 1st-degree AV block + marked sinus arrhythmia + the change in PR interval from beat #5-to-beat #6 — suggests a form of vagotonic block ( See My Comment in the October 9, 2020 post in Dr. Smith's ECG Blog ). Initial high sensitivity troponin I returned at 6ng/L (normal 0.20
The patient has active chest pain, so if these are abnormally large T-waves This link shows 13 blog posts of Posterior Reperfusion T-waves. I did not think that the T-waves in V2 and V3 are hyperacute and I still do not--I disagree with Ken below--I think they are normal , especially in the context of bradycardia.
This blog explores the ways wearable technology can help track heart health, the advantages it offers, and how it contributes to better outcomes for those requiring surgical intervention. Early detection of conditions like AFib, bradycardia, or tachycardia allows patients to address issues before they become critical.
Syncope without prodrome is a significant risk factor for cardiac syncope and poor outcome. My Comment , by K EN G RAUER, MD ( 7/5/2018 ): This blog post provides an excellent example of how a patient with SSS ( = S ick S inus S yndrome ) may present. The indication for pacemaker placement with SSS is symptomatic bradycardia.
Atrial fibrillation is also a predictor of worse outcomes in this case (Alborzi). I've copied KEY points from My Comment in the August 6, 2022 post in Dr. Smith's ECG Blog — regarding the answer to this question. Other Arrhythmias ( PACs, PVCs, AFib, Bradycardia and AV conduction disorders — potentially lethal VT/VFib ).
My most talented blog readers are paramedics because they have to put themselves on the line every time they activate the cath lab. Outcome and Analysis: ECG 1 is diagnostic of inferior and right ventricular MI. There is a junctional bradycardia. And they teach me a lot. Troponin was repeated and returned higher still.
During the night, while on telemetry, the patient became bradycardic, with periods of isorhythmic AV dissociation (nodal escape rhythm alternating with sinus bradycardia), and there were sporadic PVCs. This is what T-waves look like when there is a long QT." Below are two ECGs from the telemetry monitoring.
Smith : "What was the outcome?" Former Resident: "They took him but they rolled their eyes at me (Smith editorial comment: how often have they rolled their eyes at YOU?). They said it looked similar to his old one (in my opinion, similar, but not similar enough to be able to say no OMI)." They of course opened and stented it.
Here is his ED ECG: There is bradycardia with a junctional escape. Case continued A bedside ultrasound showed diminished LV EF and of course bradycardia. OTHER Examples of Lead Reversal on Dr. Smith's Blog: Technical errors featuring a variety of lead reversal placements remain a surprisingly common “mishap” of everyday practice.
Here is PM Cardio's Queen of Hearts interpretation (AI ECG interpretation trained by Meyers, Smith, and PM Cardio team using thousands of cases and their outcomes): The output number ranges from 0 to 1, with numbers closer to zero meaning likely NOT OMI, and numbers closer to 1 meaning OMI.
Sinus bradycardia, normal conduction, normal axis, normal R wave progression, no hypertrophy. Selected LINKS on this Topic: ECG Blog #246 — Reviews the concept of the "Mirror Test" with a clinical example. What do you think? There’s primary ST depression in the precordial leads maximal in V3-4, and an inverted T wave in V2.
Triage physician interpretation: -sinus bradycardia -lateral ST depressions While there are lateral ST depressions (V5, V6) the deepest ST depressions are in V4. Ventricular fibrillation is a common outcome in OMI and should greatly heighten the suspicion for ischemic heart disease even in the absence of profound ST elevation. =
Patient presentation is important This was a 60-something with acute chest pain: There is sinus bradycardia at a rate of 44. In case you were wondering about the T-waves and bradycardia, the K was normal. Why bradycardia? Maybe there is also inferior MI from wraparound LAD with associated sinus bradycardia.
Case outcome The Na was 109. U waves may also be found in patients with LVH and/or bradycardia , or occasionally as a normal variant. Of course this is not a difficult case because one will always measure the K in such a case, but it does show that the K is markedly affecting cardiac electrical activity.
This has been discussed many times before on this blog. In-depth discussion is beyond the scope of this blog. Opioids associate with worse outcomes in myocardial infarction , probably because they eliminate the pain signal that informs the clinician of the urgency of revascularization. Do not treat AIVR. Washam, J. Peacock, W.
Josh Kimbrell, NRP @joshkimbre Judah Kreinbrook, EMT-P @JMedic2JDoc This is the first installment of a blog series showing how transcutaneous pacing (TCP) can be difficult, and how you can improve your skills. The patient was ultimately discharged with a poor neurologic outcome.
I've been working on this a long time, thought about submitting it to a journal, but decided it gets more readers on this blog. However, none of the formulas have proven to be definitively better than another and none are well correlated with outcomes or events! This article discusses correction of the QT interval for rate.
I sent it to 5 of my OMI friends without any clinical information or outcome and all 5 independently responded with exactly the same diagnosis: "reperfused inferior OMI". MY Thoughts on the ECG in Figure-1: The rhythm in ECG #1 is sinus bradycardia at ~50-55/minute. There is ischemic ST depression in V4-V6. No chamber enlargement.
ECG#1 ECG#2 ECG#3 ECG#4 ECG#5 See outcomes of all 5 below, with the Queen of Hearts AI Bot interpretation. All of the patients presented with chest pain , and they are all in triage. Which, if any, of these patients has OMI, with myocardium at risk and need for emergent PCI? YOU TOO CAN HAVE THE PM Cardio AI BOT!!
There’s sinus bradycardia, first degree AV block, normal axis, delayed R wave progression, and normal voltages. Hyperacute T waves are deflating, suggesting reperfusion but there is still reciprocal change in I/aVL and ST depression in V2, and the bradycardia is worse. Below is the ECG. What do you think? Take home 1.
There is also bradycardia. Bradycardia puts patients at risk for "pause-dependent" Torsades de Pointes. Torsades in acquired long QT is much more likely in bradycardia because the QT interval following a long pause is longer still. The patient stabilized and had a good outcome. There is atrial fibrillation.
Like other cases on this blog who died before cath was done, I cannot prove that this patient had OMI. Could this outcome have been prevented with emergent cath? We have countless cases of missed posterior MI on this blog, including these: Interventionalist at the Receiving Hospital: "No STEMI, no cath. Were they right?
Because hyperkalemia kills by either VT or VF arrest, or by bradycardia with PEA arrest, this should be assumed to be VT until proven otherwise. While the outcome is disheartening, I think this case highlights some important management principles of a fairly common cardiac arrest phenotype. Learning Points: 1.
Cardiac Syncope ("True Syncope") Independent Predictors of Adverse Outcomes condensed from multiple studies 1. PVCs N ot generally considered abnormal ECG findings: Isolated PAC, First Degree AV Block, Sinus bradycardia at a rate of 35-45, and Nonspecific ST-T abnormalities (even if different from a previous ECG).
Theres sinus bradycardia, borderline PR interval, narrow QRS; normal axis/R wave progression; low precordial voltages, and subtle peaked T waves (most obvious in V2, but all T waves are symmetric with a narrow base). Theres no prior ECG to compare - but the bradycardia, prolonged PR and peaked T waves could all be from hyperkalemia.
Despite the baseline artifact theres sinus bradycardia, convex ST elevation in III, reciprocal ST depression in aVL and possible anterior ST depression indicating inferoposterior OMI. Prevalence and outcome of patients with non-ST segment elevation myocardial infarction with occluded culprit artery - a systemic review and meta-analysis.
The patient's long term outcome is unknown. Dr. McLaren recently wrote an excellent blog post on a similar case. As I emphasized in My Comment in the February 27, 2023 post in Dr. Smith's ECG Blog some patients may develop everything except QRS widening.
His rhythm on telemetry seemed to be sinus bradycardia vs junctional rhythm. In short with a very guarded likelihood for a positive outcome. Telemetry initially was interpreted as showing sinus bradycardia vs a junctional rhythm. A quick POCUS which showed significantly reduced ejection fraction and trace B lines.
Within ten minutes, she developed bradycardia, hypotension, and ST changes on monitor. Bradycardia and heart block are very common in RCA OMI. Was her outcome to be expected for ostial RCA OMI? There was indication of parasympathetic overdrive ( the acute inferior STEMI with profound bradycardia and junctional escape ).
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