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Background Hypertrophic cardiomyopathy (HCM) is commonly associated with atrialfibrillation (AF), but its impact on outcomes in real-world practice is uncertain. The aim of the study was to evaluate the clinical profile and prognosis of patients with HCM and AF. Methods Overall, 1739 adult patients with HCM (40.9%
Medics found him in ventricular fibrillation. He was defibrillated, but they also noticed that he was being internally defibrillated and then found that he had an implantable ICD. He was unidentified and there were no records available After 7 shocks, he was successfully defibrillated and brought to the ED.
For the past four decades, implantable cardioverter defibrillator (ICD) therapy has become the standard of care for preventing sudden cardiac death in high-risk individuals. Patients with ICDs who experience atrialfibrillation (AF) have a higher risk of stroke, heart failure, and mortality.
Primary endpoint was the incidence of patients with new onset supraventricular arrhythmia (AF, atrial flutter or any supraventricular tachycardia) lasting >30s, post PFO closure.ResultsA total of 59 patients met the inclusion criteria.
BackgroundScreening for atrialfibrillation (AF) may reveal incidental arrhythmias of relevance. Among these were sinus node dysfunction in 14 patients (1.8%), AVB in 41 (5.2%), supraventricular tachycardia in 42 (5.3%), and ventricular tachycardia in 2 (0.3%). Journal of the American Heart Association, Ahead of Print.
The team immediately paged cardiology, concerned for polymorphic ventricular tachycardia. Since sinus conducted QRS complexes cannot co-exist together with ventricular tachycardia, this must all be artifact. Such as atrialfibrillation or sinus rhythm with extrasystoles. Telemetry Sample 2 Does this change how you feel?
This can initiate ventricular arrhythmias like polymorphic ventricular tachycardia (PMVT). She was externally defibrillated with 200J and received magnesium and an IV amiodarone bolus. She was successfully defibrillated with 360J. Telemetry showed Spike-on-T phenomenon which initiated PMVT.
She was successfully revived after several rounds of ACLS including defibrillation and amiodarone. An Initial ECG was performed: Initial ECG: Sinus tachycardia with prolonged QT interval (QTc of 534 ms by Bazett). In particular — QRS alternans during narrow SVT rhythms has been associated with reentry tachycardias.
Artifact can obscure the rhythm or cause misdiagnosis of rhythms such as atrialfibrillation, ventricular tachycardia, and ventricular fibrillation. As worrisome as the initial recording in leads I and II look — a glance at lead III should immediately reassure us that defibrillation is not needed!
The rhythm now is atrialfibrillation. The arrhythmia spontaneously converted before defibrillation was achieved. A repeat ECG was recorded about 15 minutes after the initial ECG. What do you think has happened and what is the most likely diagnosis? ECG #2 Again there is a wide complex QRS due to RBBB and LAFB.
Note: Due to the limited number of normally conducted beats — it is hard to be sure whether the underlying rhythm is sinus with baseline artefact or atrialfibrillation. After resuscitation and defibrillation , there were no more episodes of TdP. Below is the patient’s 12 lead ECG following defibrillation.
It was reportedly a PEA arrest; there was no recorded V Fib and no defibrillation. The rhythm is atrialfibrillation. Tachycardia , especially in association with rapid AFib — is notorious for producing transient ST elevation not due to acute infarction ( that often resolves once heart rate slows ).
At cath, he immediately had incessant Torsades de Pointes requiring defibrillation 7 times and requiring placement of a transvenous pacer for overdrive pacing at a rate of 80. There is atrialfibrillation. See here for management of Polymorphic Ventricular Tachycardia , which includes Torsades. mEq/L: The STE is resolved.
BackgroundCatecholaminergic polymorphic ventricular tachycardia (CPVT) is a rare inherited arrhythmia disorder characterized by ventricular arrhythmia triggered by adrenergic stimulation.Case presentationA 9-year-old boy presented with convulsions following physical exertion. Genetic testing revealed a pathogenic variant of RYR2:c.720G>A
Here is a representative CXR from a different patient showing a typical CRT-D The blue dotted line overlies the right atrial lead The red dotted line overlies the RV lead. This is the shock coil and identifies this device as a defibrillator. CRT-D is cardiac resynchronization therapy with defibrillation capability, like the CXR above.
2:34 PM, following right heart catheterization She then went into atrialfibrillation with complete heart block and junctional escape rhythm prompting placement of transvenous pacemaker. Several 200 J shocks did not terminate the VF, so a second defibrillator was applied for double sequential defibrillation with 400 J.
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