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Introduction Catheter ablation is an effective and safe strategy for treating atrialfibrillation patients. Nevertheless, studies on the long-term outcomes of catheter ablation in patients with dilated cardiomyopathy are limited. Among the catheter ablation group, 58.7% ( n = 27) had persistent atrialfibrillation.
Studies have shown that mutations in the RYR2 gene, which encodes the RyR2 protein, are linked to several cardiac arrhythmias, including catecholaminergic polymorphic ventricular tachycardia (CPVT), long QT syndrome (LQTS), calcium release deficiency syndrome (CRDS), and atrialfibrillation (AF).
Catheter ablation of atrialfibrillation in infiltrative cardiomyopathies ABSTRACT Atrial and ventricular arrhythmias are common in patients with Infiltrative heart diseases. Catheter ablation is a safe and effective modality for the treatment of AF in infiltrative cardiomyopathy.
Atrialfibrillation (AF) in patients with hypertrophic obstructive cardiomyopathy (HOCM) may be caused by a primary atrial myopathy. Whether HOCM-related atrial myopathy affects mainly electrophysiological properties of the left atrium (LA) or also the right atrium (RA) has never been investigated.
The EHRA/HRS/APHRS/SOLAECE Expert Consensus on AtrialCardiomyopathies published in 2017 defined atrialcardiomyopathy as “any complex of structural, architectural, contractile, or electrophysiological changes affecting the atria.”1
Abstract Introduction In hypertrophic cardiomyopathy (HCM), atrialfibrillation (AF) has historically been regarded to have a deleterious impact on clinical course, strongly associated with progressive heart failure (HF) symptoms. Results Among the 218 patients, 50 (23%) had a history of AF and comprise the primary study cohort.
ABSTRACT Background Atrialfibrillation and atrial flutter are relatively rare in young people and the incidence of thromboembolic complications is unknown. Structural heart disease (SHD) was present in 120 patients (57%) and 20 patients (10%) had a primary cardiomyopathy.
Circulation: Arrhythmia and Electrophysiology, Ahead of Print. Atrialcardiomyopathy is a condition that causes electrical and contractile dysfunction of the atria, often along with structural and functional changes.
Circulation: Arrhythmia and Electrophysiology, Volume 16, Issue 11 , Page e012191, November 1, 2023. BACKGROUND:The cause of hypertrophic cardiomyopathy (HCM) in the young is highly varied. Atrioventricular AP (true AP) were distinguished from fasciculoventricular fibers (FVF) using standard invasive electrophysiology study criteria.
ABSTRACT Atrialfibrillation (AF) is the most common cause of arrhythmia-induced cardiomyopathy. Effective management strategies include medical therapy for rate and rhythm control, catheter ablation (CA), and goal-directed medical therapy.
Atrialcardiomyopathy represents structural and electrophysiologic abnormalities associated with atrialfibrillation (AF). Voltage data is readily acquired in an ablation procedure but comparing voltage between subjects is complicated by variation in atrial size and orientation.
The advent of transseptal puncture has enabled the study of atrialfibrillation mechanisms, while epicardial access has enabled exploration of the epicardium in ventricular arrhythmias (ventricular tachycardia/ventricular fibrillation [VF]).1,2
Complete left bundle branch block (CLBBB)-like QRS morphology of right ventricular pacing at pacemaker implantation satisfying the American Heart Association/American College of Cardiology Foundation/Heart Rhythm Society criteria of CLBBB was associated with development of pacing induced cardiomyopathy.
Within the last six months, separate AI-ECG algorithms for detecting Low Ejection Fraction (Anumana), Hypertrophic cardiomyopathy (Viz.ai), and Occlusion Myocardial infarction (Powerful Medical) have all been granted regulatory clearance (the latter under the EU MDR) and are in the early stages of deployment.
24 will focus on the following three current guideline updates: American College of Cardiology (ACC)/American Heart Association (AHA) Guidelines 2023 AtrialFibrillation Guideline - Pharmacology II: Strokes vs. Bleeds, What Do the Guidelines Tell Us About Practical Management in A-fib? The Guidelines Sessions at ACC.24 12:15 p.m.
She also has a hx of paroxysmal atrialfibrillation and is on oral anticoagulant treatment. There is almost always the possibility of exceptions ( ie, prior infarction or cardiomyopathy that might result in a very abnormal baseline tracing ). The last echocardiography 12 months ago showed HFmrEF.
Institutional Coronary Artery Bypass Case Volumes and Outcomes European Journal of Heart Failure October 2023 Makoto Mori Robotic Mitral Valve Repair for Degenerative Mitral Regurgitation The Annals of Thoracic Surgery August 2023 Carlos Diaz-Castrillion Volume-Failure to Rescue Relationship in Acute Type A Aortic Dissections: An Analysis of The Society (..)
Institutional Coronary Artery Bypass Case Volumes and Outcomes European Journal of Heart Failure October 2023 Makoto Mori 1 Robotic Mitral Valve Repair for Degenerative Mitral Regurgitation The Annals of Thoracic Surgery August 2023 Carlos Diaz-Castrillion 2 Volume-Failure to Rescue Relationship in Acute Type A Aortic Dissections: An Analysis of The (..)
The absence of any wall motion abnormality makes ischemic cardiomyopathy very unlikely. Patient course The patient was started on beta blockers and schedule for an electrophysiologic study. The new onset cardiomyopathy was thought to be due to both drug/alcohol use and to Tachycardia-Induced Cardiomyopathy.
Atrail electroanatomic voltage mapping (Atrial-EAVM) and late gadolinium enhancement imaging (Atrial-LGE) quantify the functional and anatomic extent of atrial fibrosis, a central component of atrialcardiomyopathy. Atrial-EAVM and Atrial-LGE correlated with greatest agreement at high-fibrosis burdens.
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