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Here was his initial ED ECG: There is atrialfibrillation with a rapid ventricular response. ST depression is common BOTH after resuscitation from cardiacarrest and during atrial fib with RVR. and repeat the ECG, to see if the apparent ischemia persists. He had a history of CAD with CABG. This was done.
The patient had a combined respiratory and metabolic acidosis (as we commonly find in those with prolonged arrest), and a K of 4.1, The rhythm is nearly regular, but there are no P-waves (it is too regular to be atrialfibrillation). Was this: 1) ACS with ischemia and spontaneous reperfusion? at the time of the ECG.
Written by Pendell Meyers First try to interpret this ECG with no clinical context: The ECG shows an irregularly irregular rhythm, therefore almost certainly atrialfibrillation. After an initially narrow QRS, there is a very large abnormal extra wave at the end of the QRS complex. There is also large T wave inversion and long QT.
Edited by Bracey, Meyers, Grauer, and Smith A 50-something-year-old female with a history of an unknown personality disorder and alcohol use disorder arrived via EMS following cardiacarrest with return of spontaneous circulation. Cardiac Motion Alternans — is the result of cardiac movement rather than electrical alternation.
Case submitted and written by Mazen El-Baba MD, with edits from Jesse McLaren and edits/comments by Smith and Grauer A 90-year old with a past medical history of atrialfibrillation, type-2 diabetes, hypertension, dyslipidemia, presented with acute onset chest/epigastric pain, nausea, and vomiting. Anything more on history?
Alternation in ST segment appearance ( or in the amount of ST elevation or depression ) — is often linked to ischemia. Conduction and refractoriness alternans may be seen with WPW-related as well as AV Nodal-dependent reentr y tachycardias — atrialfibrillation — acute pulmonary embolus — myocardial contusion — and severe LV dysfunction.
The rhythm is atrialfibrillation. In terms of ischemia, there is both a signal of subendocardial ischemia (STD max in V5-V6 with reciprocal STE in aVR) AND a signal of transmural infarction of the inferior wall with Q wave and STE in lead III with reciprocal STD in I and aVL. The QRS complex is within normal limits.
The first task when assessing a wide complex QRS for ischemia is to identify the end of the QRS. The ST segment changes are compatible with severe subendocardial ischemia which can be caused by type I MI from ACS or potentially from type II MI (non-obstructive coronary artery disease with supply/demand mismatch). What do you think?
Remember, in diffuse subendocardial ischemia with widespread ST-depression there may b e ST-E in lead s aVR and V1. There are well formed R-waves with good voltage/amplitude which is uncommon for ischemia. Smith: This bizarre ECG looks like a post cardiacarrest ECG with probable acidosis or hyperkalemia in addition to OMI.
There is atrialfibrillation. Comments: STEMI with hypokalemia, especially with a long QT, puts the patient at very high risk of Torsades or Ventricular fibrillation (see many references, with abstracts, below). If cardiacarrest from hypokalemia is imminent (i.e., mEq/L: The STE is resolved. mEq/L, from 1.9
AtrialFibrillationAtrialfibrillation causes irregular heartbeat, and the heart's normal blood supply is affected. Since atrialfibrillation can also be intermittent, such patients should continuously monitor their heart activity while performing daily activities with a portable ECG device.
They include myocardial ischemia, acute pericarditis, pulmonary embolism, external compression due to mass over the right ventricular outflow tract region, and metabolic disorders like hyper or hypokalemia and hypercalcemia. Atrialfibrillation and Brugada syndrome. mV or R/q ≥ 0.75. Heart Rhythm. 2016 Oct;13(10):e295-324.
Osborn waves have been reported with hypercalcemia, brain injury, subarachnoid hemorrhage, Brugada syndrome, cardiacarrest from VFib — and — severe, acute ischemia resulting in acute MI ( See My Comment in the November 22, 2019 post on Dr. Smith’s Blog ). Rituparna et al — as well as Chauhan and Brahma ( Int.
Occurrence of “J Waves” in 12-Lead ECG as a Marker of Acute Ischemia and Their Cellular Basis. Occurrence of "J waves" in 12-lead ECG as a marker of acute ischemia and their cellular basis. The final letter in the SLOWED mnemonic is " D " for "Dead" ( resulting from VT/VF or asystolic cardiacarrest ).
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