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Medics found him in ventricular fibrillation. He was defibrillated, but they also noticed that he was being internally defibrillated and then found that he had an implantable ICD. He was unidentified and there were no records available After 7 shocks, he was successfully defibrillated and brought to the ED.
Here was his initial ED ECG: There is atrialfibrillation with a rapid ventricular response. ST depression is common BOTH after resuscitation from cardiacarrest and during atrial fib with RVR. Not all patients with ventricular fibrillation necessarily need emergent angiography. The patient was cardioverted.
While on telemetry monitoring he suffered cardiacarrest and was resuscitated. What ECG finding may have contributed to (or precipitated) the cardiacarrest? After resuscitation and defibrillation , there were no more episodes of TdP. Below is the patient’s 12 lead ECG following defibrillation.
Rate vs Rhythm Control in AtrialFibrillation Rate vs rhythm control as a management strategy in atrialfibrillation has been a long standing topic for debate. EAST-AFNET 4 trial had 2789 patients with early atrialfibrillation and cardiovascular conditions [8]. years of follow up per patient. N Engl J Med.
She was found to be in ventricular fibrillation and was defibrillated 8 times without a single, even transient, conversion out of fibrillation. Fine ventricular fibrillation She received 2 mg epinephrine, 150 mg amiodarone and underwent chest compressions with the LUCAS device. at the time of the ECG. Mg was 1.6.
It was reportedly a PEA arrest; there was no recorded V Fib and no defibrillation. The rhythm is atrialfibrillation. He had multiple cardiacarrests with ROSC regained each time. This patient arrested shortly after hospital arrival. The QRS complex is within normal limits.
The rhythm now is atrialfibrillation. The arrhythmia spontaneously converted before defibrillation was achieved. This patient is actively dying from a left main coronary artery OMI and cardiacarrest from VT/VF or PEA is imminent! Complete LMCA occlusion is associated with clinical shock and/or cardiacarrest.
Edited by Bracey, Meyers, Grauer, and Smith A 50-something-year-old female with a history of an unknown personality disorder and alcohol use disorder arrived via EMS following cardiacarrest with return of spontaneous circulation. She was successfully revived after several rounds of ACLS including defibrillation and amiodarone.
Smith’s ECG Blog: SQTS is an inherited cardiac channelopathy determined by the presence of symptoms ( syncope, cardiacarrest ) — positive family history — and the ECG finding of an abnormally short QTc interval. Treatment is by ICD ( implantable cardioverter defibrillator ).
At cath, he immediately had incessant Torsades de Pointes requiring defibrillation 7 times and requiring placement of a transvenous pacer for overdrive pacing at a rate of 80. There is atrialfibrillation. If cardiacarrest from hypokalemia is imminent (i.e., After pacing, there was no recurrence of Torsades.
The patient was put on Extracorporeal Life Support in the ED 3 hours after initial resuscitation, the core temp was 30° C and the patient was defibrillated with a single attempt. Perhaps the bifascicular block ( RBBB/LPHB ) present on ECG #1 — but which resolved by ECG #2 — was also ischemic-related from the cardiacarrest.
Smith’s ECG Blog: SQTS is an inherited cardiac channelopathy determined by the presence of symptoms ( syncope, cardiacarrest ) — positive family history — and the ECG finding of an abnormally short QTc interval. Treatment is by ICD ( implantable cardioverter defibrillator ).
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