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MILLER, PHD, LED THE DEVELOPMENT OF A NEW "ATHEROSCLEROSIS ATLAS" THAT DETAILS, AT THE LEVEL OF INDIVIDUAL CELLS, CRITICAL PROCESSES RESPONSIBLE FOR FORMING THE HARMFUL PLAQUE BUILDUP THAT CAUSES HEART ATTACKS, STROKES AND CORONARYARTERYDISEASE. THE UNIVERSITY OF VIRGINIA SCHOOL OF MEDICINE'S CLINT L.
The triglyceride-glucose (TyG) index is considered a dependable biomarker for gauging insulin resistance. The atherogenic index of plasma (AIP) represents a marker reflecting atherosclerosis. However, there is.
Atherosclerotic cardiovascular disease (ASCVD), caused by plaque buildup in arterial walls, is one of the leading causes of disability and death worldwide.1,2 1,2 ASCVD causes or contributes to conditions that include coronaryarterydisease (CAD), cerebrovascular disease, and peripheral vascular disease (inclusive of aortic aneurysm).3
ObjectiveThe present study aimed at evaluating the association between sympathetic nervous system activation (SNS) and the severity of coronaryarterydisease (CAD).
Atherosclerotic coronaryarterydisease (CAD) is the causal pathological process driving most major adverse cardiovascular events (MACE) worldwide. The complex development of atherosclerosis manifests as intimal plaque which occurs in the presence or absence of traditional risk factors.
Coronaryartery calcification (CAC) accompanies the development of advanced atherosclerosis. Nevertheless, the relationship between CAC and the susceptibility of a plaque to provoke a thrombotic event remains incompletely understood. This review summarizes the current understanding and literature on CAC.
Introduction:Inflammation plays an important role in the pathogenesis of coronaryarterydisease and Acute Coronary Syndrome (ACS). Conclusion:The SIRI and SII are inflammatory biomarkers that should be studied as potentially predictors of MACE among patients presented with ACS.
An elevated Lp(a) is a common genetic factor that is independently and causally related to premature coronaryarterydisease. In my experience, some patients present later in life with a significantly elevated Lp(a) but have no evidence of coronaryarterydisease. Atherosclerosis. 2017 Jan;256:47-52.
Share For those with defective LDL receptors, as seen in familial hyperlipidemia, APOB particles are not cleared effectively, and concentrations rise, causing atherosclerosis 4. Which increases the risk of atherosclerosis. Identification of a new plasma biomarker of Alzheimer's disease using metabolomics technology.
They are also at an increased risk of coronaryarterydisease (Discussed in more detail here ). Additionally, access to cholesterol synthesis biomarker testing, including desmosterol and lathosterol levels, can be challenging to obtain. Atherosclerosis. This one requires some nuance. Curr Opin Lipidol. J Lipid Res.
9 Adults who sleep <6 hours each night have a 15% greater risk for CVD and 23% increased risk for coronary heart disease compared to those sleeping 7 hours or more. 12 Sleep duration is not only associated with diseases of the cardiovascular system, but also with direct biological markers of CVD risk.
Coronaryartery calcium (CAC) is a measure of atherosclerosis and a strong predictor of cardiovascular disease. All athletes—regardless of predisposition to heart disease—should be prudent in following a generally healthy lifestyle. A new analysis helps us understand.
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