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A small proportion of patients with STEMI treated via primary PCI experienced late ventricular tachycardia (VT) or ventricular fibrillation (VF), occurring one or more days following the procedure, but late VT or VF with cardiacarrest occurred rarely, especially among patients with uncomplicated STEMI, according to a study published in JAMA Network (..)
The 2019 ESC Guidelines for the management of patients with supraventricular tachycardia indicated that IV Amiodarone should not be considered in these populations. Regarding AFib with WPW: The very rapid heart rate and at times extremely short R-R intervals put the patient with AFib and WPW at risk of cardiacarrest from VFib.
Here was his initial ED ECG: There is sinus tachycardia at a rate of about 140 There is profound ST Elevation across all precordial leads, as well as I and aVL. If a patient presents with chest pain and a normal heart rate, or with shockable cardiacarrest, then ischemic appearing ST elevation is STEMI until proven otherwise.
Here is the ECG: Sinus tachycardia. If cardiacarrest from hypokalemia is imminent (i.e., CASE : Prehospital CardiacArrest due to Hypokalemia I recently had a case of prehospital cardiacarrest that turned out to be due to hypokalemia. This patient presented with severe DKA. What do you think?
Is it ventricular tachycardia (VT) due to hyperK or is it a supraventricular rhythm with hyperK? Here are other posts on hyperK, large calcium doses for hyperK, and ventricular tachycardia in hyperK Weakness, prolonged PR interval, wide complex, ventricular tachycardia Very Wide and Very Fast, What is it? How would you treat?
These include ( among others ) — acute febrile illness — variations in autonomic tone — hypothermia — ischemia-infarction — malignant arrhythmias — cardiacarrest — and especially Hyperkalemia. Sinus Tachycardia ( common in any trauma patient. ).
And the article was rejected. MY Thoughts on ECG #1: The rhythm is sinus tachycardia at 105-110/minute. The cardiology reviewers said that "No one strictly follows STEMI criteria. The 4th universal definition mentions ST depression, posterior MI, and T-wave changes." The PR and QRS intervals are normal.
4 Unfortunately, this article provides no electrocardiographic, echo, or angiographic data, so it is not certain that these high levels were in the absence of acute MI. This sinus tachycardia ( at ~130/minute ) — is consistent with the patient’s worsening clinical condition, with development of cardiogenic shock.
See here for management of Polymorphic Ventricular Tachycardia , which includes Torsades. If cardiacarrest from hypokalemia is imminent (i.e., However, this review references the Sterns article above, which by my reading does not state this. Could the dysrhythmias have been prevented? mEq/L, from 1.9
This progressed to electrical storm , with incessant PolyMorphic Ventricular Tachycardia ( PMVT ) and recurrent episodes of Ventricular Fibrillation ( VFib ). baseline (this is what most recommend but seems like far too much QRS widening to me) = See these articles and this graphic: 1.
This article discusses correction of the QT interval for rate. The article is written by Dr. Smith and Dr. Friedman. Answer : you must treat the patient's underlying condition causing sinus tachycardia, and repeat the ECG at the lower heart rate. In that article, they do not say what is a dangerously short QT is (e.g.
Josep Brugada way back in 2001 for the inaugural issue of IPEJ, along with his review article. Brugada’s article was the first ever article which I received for IPEJ and it gave a great boost to the debut issue of the journal [1]. I am always happy to see this ECG of Brugada syndrome as it was sent to me by Prof.
After initiating treatment for hyperkalemia, repeat ECG showed resolution of Brugada pattern: The ECG shows sinus tachycardia. Steve, what do you think of this ECG in this CardiacArrest Patient?" A woman in her 50s with dyspnea and bradycardia A patient with cardiacarrest, ROSC, and right bundle branch block (RBBB).
The article is edited by Smith. Otherwise vitals after intubation were only notable for tachycardia. An initial EKG was obtained: Computer read: sinus tachycardia, early acute anterior infarct. This was submitted by Alexandra Schick. Dr. Schick is a PGY3 at the Brown Emergency Medicine Residency in Rhode Island.
There is sinus tachycardia and also a large R-wave in aVR. Drug toxicity , especially diphenhydramine , which has sodium channel blocking effects, and also anticholinergic effects which may result in sinus tachycardia, hyperthermia, delirium, and dry skin. Her temperature was 106 degrees. As part of the workup, she underwent an ECG.
We showed this in this article in JAMA Cardiology. A retrospective 'target trial emulation' comparing amiodarone and lidocaine for adult out-of-hospital cardiacarrest resuscitation. The final angiographic result is very good. Troponins Initial troponin was 24 ng/L (barely above URL). DOI: 10.1016/j.resuscitation.2025.110515
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