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The increased risk of arrhythmias and suddencardiacdeath (SCD) caused by drug-induced QT prolongation has been a prominent safety pharmacology concern for the last 30 years.
Concerns regarding the risk of cardiacarrhythmia and death have been raised, yet the burden of HCQ and CQ-related cardiac toxicities remains unclear. Concerns regarding the risk of cardiacarrhythmia and death have been raised, yet the burden of HCQ and CQ-related cardiac toxicities remains unclear.
Contemporary antiarrhythmic treatment of Timothy syndrome 1 (TS1) is inadequate to prevent suddencardiacdeath. Using electroanatomic mapping (EAM) we discovered cardiac activation delay, which creates the milieu for functional reentrant ventricular arrhythmias.
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