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Mutations in RyR2 and its dysfunction are implicated in various congenitalheartdiseases (CHDs). ABSTRACT Ryanodine receptor 2 (RyR2) protein, a calcium ion release channel in the sarcoplasmic reticulum (SR) of myocardial cells, plays a crucial role in regulating cardiac systolic and diastolic functions.
ABSTRACT Introduction Data regarding safety and long-term outcome of very high-power-short duration (vHPSD) ablation in adult congenitalheartdisease (ACHD) patients with paroxysmal or persistent atrial fibrillation (AF) are lacking. Methods Retrospective observational single-center study. with paroxysmal AF, 43.6%
All these features together in a cyanotic congenitalheartdisease is characteristic of tricuspid atresia. Fragmented QRS is a marker of myocardial scar and consequent arrhythmias in ischemic and nonischemic cardiomyopathy.
A 37-year-old woman with biventricular repair for pulmonary atresia and an intact ventricular septum was referred for an electrophysiological study in the context of recurrent atrial arrhythmias with multiple electrical cardioversions. Her clinical tachycardia was easily inducible and had a cycle length (TCL) of 340 ms.
Atrial conduction abnormalities are common among patients with congenitalheartdisease, predisposing them to arrhythmias. In clinical practice, substrates are usually identified during electrophysiological mapping procedures using bipolar electrograms (EGM).
Abstract Introduction Management of transvenous leads in patients with congenitalheartdisease (CHD) can be complicated by venous obstructions and residual shunts. Complications included blood transfusion ( n = 2), arrhythmia ( n = 3), pleural effusion ( n = 1), and pressure ulcer ( n = 1). h (range 5.4−12.8 h).
Transcript of the video: Ebstein’s Anomaly is one of the cyanotic congenitalheartdisease in which survival to adult life is common. This can be a source of cardiac arrhythmia as well. Electrophysiological study will show that, and this pathway can be ablated.
Circulation: Arrhythmia and Electrophysiology, Ahead of Print. BACKGROUND:A growing number of patients with tetralogy of Fallot develop left ventricular systolic dysfunction and heart failure, in addition to right ventricular dysfunction.
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