This site uses cookies to improve your experience. To help us insure we adhere to various privacy regulations, please select your country/region of residence. If you do not select a country, we will assume you are from the United States. Select your Cookie Settings or view our Privacy Policy and Terms of Use.
Cookie Settings
Cookies and similar technologies are used on this website for proper function of the website, for tracking performance analytics and for marketing purposes. We and some of our third-party providers may use cookie data for various purposes. Please review the cookie settings below and choose your preference.
Used for the proper function of the website
Used for monitoring website traffic and interactions
Cookie Settings
Cookies and similar technologies are used on this website for proper function of the website, for tracking performance analytics and for marketing purposes. We and some of our third-party providers may use cookie data for various purposes. Please review the cookie settings below and choose your preference.
Strictly Necessary: Used for the proper function of the website
Performance/Analytics: Used for monitoring website traffic and interactions
In adults with congenital heart disease (ACHD patients), atrial arrhythmias (AA) and heart failure (HF) are common. 1 Factors related to congenital heart disease, such as underlying anatomy, surgical repair technique and scars, can all be considered as AA substrates. In the current issue, Lauwers and coworkers 3 set out.
Background Atrial arrhythmias (AA) and heart failure (HF) are major causes of hospitalisation in adult congenital heart disease (ACHD). Methods In this single-centre retrospective cohort study, data from 3995 patients with ACHD were analysed.
The prevalence of congenital heart disease (CHD) in adult patients has risen with advances in diagnostic and surgical techniques. Surgical modifications and hemodynamic changes increase the susceptibility to arrhythmias, impacting morbidity and mortality rates, with arrhythmias being the leading cause of hospitalizations and sudden deaths.
Methods We included adult patients who had undergone Fontan surgery for congenital heart disease and were pregnant between 1994 and 2021. We aimed to evaluate the immediate and long-term outcomes among these patients who were followed at our centre. We examined maternal and obstetric outcomes.
Is engagement in vigorous exercise, including competitive sports, associated with increased risk for life-threatening ventricular arrhythmias in individuals with known and treated congenital long-QT syndrome (LQTS)?
There is limited data comparing arrhythmia burden amongst patients with congenitally corrected transposition of the great arteries (cc-TGA) undergoing anatomic repair (AR), physiologic repair (PR), and non-surgical management (NS).
Atrial arrhythmias are an important source of morbidity in adults with congenital heart disease (CHD) and have been linked to sudden death in certain subgroups. Whereas intra-atrial reentrant tachycardia (IART) is the most common arrhythmia, the prevalence of atrial fibrillation (AF) is increasing.
Transcatheter pulmonary valve replacement (TPVR) has become a safe and effective alternative to surgical PVR in tetralogy of Fallot (TOF), isolated pulmonary stenosis (PS), and other congenital heart disease (CHD) variants.
Congenital long QT syndrome (LQTS) is characterized by delayed ventricular repolarization, predisposing to potentially lethal ventricular arrhythmias. The variability in disease severity among patients remains largely unexplored, underscoring the limitations of current risk stratification methods.
IntroductionVentricular septal defect (VSD) is a common congenital heart disease (CHD), accounting for 2030% of all CHD cases. Minor complications included transient arrhythmias (6.2%), femoral hematoma (8.6%), and mild allergic reactions to contrast agents (9.9%). Data were analyzed using SPSS 20.0.ResultsThe
Patients with adult congenital heart disease (ACHD) such as Tetralogy of Fallot (ToF) are at high risk for ventricular arrhythmias requiring ablation. Less common syndromes such as double outlet right ventricle (DORV) and Williams Syndrome provide additional complications.
In this weeks View, Dr. Eagle looks at what adult cardiologists should know about congenital heart disease patients. He then discusses sudden cardiac arrest and death in competitive and recreational athletes.
Introduction Heart transplantation (HT) is the only treatment option in children with heart failure secondary to cardiomyopathies and non-reparable congenital heart diseases. 111.87), arrhythmias were detected in 9 (33%) patients. We did not identify significant risk factors for arrhythmias post-HT. On a median follow-up of 35.07
Objectives Patients with congenital heart disease (CHD) are increasingly pursuing pregnancy, highlighting the need for data on late cardiovascular events (more than 6 months after delivery). Late cardiovascular events were defined as heart failure, arrhythmia, thromboembolic events, endocarditis, urgent cardiovascular interventions or death.
Congenital heart disease is a daunting diagnosis for any parent. We’ll cover common types of medications used to treat congenital heart disease, what parents should know about their use, and potential side effects to watch out for. Be sure to talk to your doctor if you have any questions or concerns.
Despite improved childhood survival in congenital heart disease (CHD) due to advances in management, late-onset sudden cardiac death (SCD) from malignant ventricular arrhythmias remains a leading cause of mortality in adults with CHD (ACHD).
Mutations in RyR2 and its dysfunction are implicated in various congenital heart diseases (CHDs). ABSTRACT Ryanodine receptor 2 (RyR2) protein, a calcium ion release channel in the sarcoplasmic reticulum (SR) of myocardial cells, plays a crucial role in regulating cardiac systolic and diastolic functions.
Causes of Cardiac Arrest Cardiac arrest can be triggered by various factors, including: Abnormal heart rhythms (arrhythmias) Severe coronary artery disease High blood pressure Extreme electrolyte imbalances Drug overdose or trauma Congenital heart defects Recognizing Sudden Cardiac Arrest Unlike a heart attack, sudden cardiac arrest occurs without (..)
ABSTRACT Introduction Data regarding safety and long-term outcome of very high-power-short duration (vHPSD) ablation in adult congenital heart disease (ACHD) patients with paroxysmal or persistent atrial fibrillation (AF) are lacking. Methods Retrospective observational single-center study. with paroxysmal AF, 43.6% with persistent AF).
All these features together in a cyanotic congenital heart disease is characteristic of tricuspid atresia. Fragmented QRS is a marker of myocardial scar and consequent arrhythmias in ischemic and nonischemic cardiomyopathy.
Methods Patients from the Swiss Adult Congenital HEart disease Registry were included. Predictors of a composite adverse event (arrhythmias, hospitalisation for heart failure and death) were identified by multivariate Cox regression analysis. NT-proBNP levels, echocardiography, exercise testing and MRI data were collected.
Abstract Introduction We report the case of an 18-year-old female with recurrent syncope that was discovered to have congenital long QT syndrome (LQTS) and episodes of a transiently short QT interval after spontaneous termination of polymorphic ventricular tachycardia.
Evaluation of escape rates and ventricular ectopy with exercise in complete heart block is an important aspect in the evaluation of congenital complete heart block. ECG showing congenital complete heart block with ventricular rate of 47/min and atrial rate of 63/min. Tracing shows bidirectional ventricular ectopics.
Two articles and an accompanying editorial in Heart Rhythm , the official journal of the Heart Rhythm Society, the Cardiac Electrophysiology Society , and the Pediatric & Congenital Electrophysiology Society , published by Elsevier , address the controversy and recommend shortening the blanking period.
Abstract Introduction Management of transvenous leads in patients with congenital heart disease (CHD) can be complicated by venous obstructions and residual shunts. Complications included blood transfusion ( n = 2), arrhythmia ( n = 3), pleural effusion ( n = 1), and pressure ulcer ( n = 1). Median procedure time was 9.8 h
The institute specializes in the treatment of coronary artery diseases, arrhythmia, advanced heart failure and heart transplantation, structural heart disease, cardiogenetics, and congenital heart disease. Additionally, it offers comprehensive cardiac screening and preventive care.
Patients with dextro-transposition of the great arteries (d-TGA) are at risk for recurrent atrial arrhythmias late after atrial switch operations. Advanced anatomic knowledge including prior surgical incisions are integral in planning the ablation procedure.
Tetralogy of Fallot (TOF) is the most prevalent form of cyanotic congenital heart disease and has become the poster child for the evolution and success of neonatal cardiac surgery.
A 37-year-old woman with biventricular repair for pulmonary atresia and an intact ventricular septum was referred for an electrophysiological study in the context of recurrent atrial arrhythmias with multiple electrical cardioversions. Her clinical tachycardia was easily inducible and had a cycle length (TCL) of 340 ms.
It should be kept in mind that on occasions, beta-one agonist can result in increased ventricular ectopy e.g., in severe myocardial ischemia (by increasing myocardial demand), or sometimes with congenital long-QT syndrome. Therefore, I usually prefer temporary pacing which might be more controlled and is more predictable."
Atrial conduction abnormalities are common among patients with congenital heart disease, predisposing them to arrhythmias. In clinical practice, substrates are usually identified during electrophysiological mapping procedures using bipolar electrograms (EGM).
Arrhythmias (Irregular Heartbeats) Persistent abnormal heart rhythms can disrupt the heart’s pumping efficiency, eventually causing it to enlarge to maintain blood flow. Congenital Heart Defects Some individuals are born with heart defects that cause parts of the heart to work harder, leading to enlargement over time.
the primary cardiac Nav α-subunit, has been extensively implicated in the etiology of arrhythmias. due to congenital or acquired defects have been associated with a wide range of pathologic manifestations, including sinus nodal dysfunction, atrial and ventricular arrhythmias, and heart failure. Indeed, dysfunctions in Nav1.5
Congenital Long QT Syndrome (LQTS) can result in fatal cardiac arrhythmias. The main pharmacological treatment includes beta-blockers; however, while effective at reducing cardiac events, beta-blockers do not treat underlying mechanisms of the disease, and patients occasionally experience breakthrough cardiac events or side effects.
Congenital Long QT Syndrome (LQTS) can result in fatal cardiac arrhythmias. The main pharmacological treatment includes beta-blockers; however, while effective at reducing cardiac events, beta-blockers do not treat underlying mechanisms of the disease, and patients occasionally experience breakthrough cardiac events or side effects.
Transcript of the video: Ebstein’s Anomaly is one of the cyanotic congenital heart disease in which survival to adult life is common. This can be a source of cardiac arrhythmia as well. An important cause of arrhythmias in Ebstein’s anomaly is the presence of a right sided accessory atrioventricular pathway or WPW syndrome.
Atrial septal defect (ASD) is a congenital heart defect. Known as one of the most common congenital heart defects in children, ASD affects more than 2,000 babies each year. Preventing ASD Since ASD is a congenital heart condition that develops in the fetus and is present at birth, it’s not possible to prevent it.
Ventricular tachycardia is a potentially life threatening cardiac arrhythmia. Classical example of polymorphic ventricular tachycardia is torsade de pointes associated with congenital or acquired QT interval prolongation. Beta blockers are needed in addition, for most persons with congenital long QT syndrome.
In the setting of arrhythmias, specific SDoHs can increase the incidence of atrial fibrillation and adversely affect major outcomes in these patients. In congenital heart diseases, SDoHs can affect major outcomes, as well.
In preparation for the ABIM Cardiovascular Disease exam, check out the BoardVitals Cardiology Board Review Question Bank and we’ll make sure you’re well versed in the following 13 areas covered on the exam: Multiple-Choice Component Arrhythmias 15% Coronary Artery Disease 23% Heart Failure and Cardiomyopathy 17% Valvular Disease 15% Pericardial (..)
T-wave alternans and the susceptibility to ventricular arrhythmias. Chronic amiodarone evokes no torsade de pointes arrhythmias despite QT lengthening in an animal model of acquired long-QT syndrome. Both ST segment and T wave alternans have been known to precede malignant ventricular arrhythmias. Pacing Clin Electrophysiol.
Circulation: Arrhythmia and Electrophysiology, Ahead of Print. BACKGROUND:A growing number of patients with tetralogy of Fallot develop left ventricular systolic dysfunction and heart failure, in addition to right ventricular dysfunction.
We organize all of the trending information in your field so you don't have to. Join thousands of users and stay up to date on the latest articles your peers are reading.
You know about us, now we want to get to know you!
Let's personalize your content
Let's get even more personalized
We recognize your account from another site in our network, please click 'Send Email' below to continue with verifying your account and setting a password.
Let's personalize your content