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Clinical introduction A woman in her 60s with non-obstructive coronary artery disease, aortic valve replacement and aortic arch repair, chronic diastolic heart failure and paroxysmal atrial fibrillation (AF) and flutter (AFL), presented with 3 days of sustained palpitations that felt similar to prior episodes of AF/AFL.
Due to the chest pain radiating into the patient's back, the ER physician ordered CTA chest to rule out aortic dissection. The constellation of dyspnea, tachycardia, and (relatively) low voltage on ECG should prompt immediate evaluation for pericardial effusion and tamponade.
There is limited data regarding safety, electrophysiologic characteristics and arrhythmia substrate during ventricular tachycardia (VT) ablation in patients with prior aortic valve replacement (AVR).
We present a patient with a history of heart failure and metallic aortic and mitral valves surgeries, who required ablation for a drug-refractory left ventricular tachycardia. But the metallic valves prohibite.
Abstract Introduction Catheter-based radiofrequency (RF) ablation is generally regarded as the standard approach for patients with ventricular tachycardia (VT) refractory to antiarrhythmic drug therapy and may be considered as a first-line approach when there is a preference to avoid these agents.
Sinus tachycardia has many potential causes. This is especially true for the elderly patient with sinus tachycardia. What is the cause of the sudden tachycardia? She had a very elevated troponin T at 12,335 ng/L at the time of presentation. The patient in today’s case suddenly became tachycardic while sleeping.
He developed atrial tachycardia (AT) and underwent catheter ablation. AT was diagnosed as peri-mitral flutter and the mitral isthmus (MI) linear ablation via a trans-aortic approach successfully terminated it.
The ECG shows sinus tachycardia with RBBB and LAFB, without clear additional superimposed signs of ischemia. The Initial ECG in Today's Case: As per Dr. Meyers — the initial ECG in today's case shows sinus tachycardia with bifascicular block ( = RBBB/LAHB ). Sinus Tachycardia ( common in any trauma patient. ).
An increase in myocardial oxygen demand due to tachycardia, elevated ventricular afterload (BP or Aortic stenosis), increased wall stretch, or a decrease in oxygen supply due to hypotension, anemia, hypoxia, sepsis, or a combination of all of the above. Type II ischemia.
EMS reports intermittent sinus tachycardia and bradycardia secondary to some type of heart block during transport. It is hard to make out P waves but you can see them best in V2, and notches in the T waves in other leads - this is a sinus tachycardia with a very long PR interval indicating first degree block.
Post PCI an intra-aortic balloon pump (IABP) was placed and a combination of norepinephrine and dobutamine was needed to maintain perfusion pressures. The image quality is not the best, but you can see the subtotal occlusion of the left main artery. Below is a still image with the red arrow indicating the subtotal LMCA stenosis.
We report our initial experience in using this catheter for the treatment of outflow premature ventricular contractions (PVCs) or repetitive non-sustained monomorphic ventricular tachycardias (VTs). The DTA parameters were adjusted to a target-temperature of 60C with 50W power.
AS-APs can be successfully ablated from the right atrium (RA) or the aortic valve's noncoronary cusp (NCC). Methods and Results A 21-year-old female with supraventricular tachycardia (SVT) and pre-excitation on electrocardiogram (ECG) underwent electrophysiology study (EPS) confirming an AS-AP with anterograde and retrograde conduction.
Look at the aortic outflow tract. The diagnostic coronary angiogram identified only minimal coronary artery disease, but there was a severely calcified, ‘immobile’ aortic valve. Aortic angiogram did not reveal aortic dissection. In fact, bedside ultrasound might even find severe aortic stenosis. What do you see?
Discover 2024’s Critical Advances in Cardiometabolic Care Here are some recent advances in cardiology: Aortic Disease : Moderate hypothermia during aortic arch surgery is effective, reducing the need for deep hypothermia.
The next morning the patient went for his routine echocardiogram, where the operator noticed a dilated aortic root at 5.47 cm with severe aortic insufficiency. The team was notified and they ordered a stat aortagram which showed type A aortic dissection from the aortic valve to the iliacs.
Category 2 : An increase in myocardial oxygen demand due to tachycardia, elevated ventricular afterload (BP or aortic stenosis), or increased wall stretch (admittedly this latter is more complicated) or a decrease in oxygen supply due to hypotension, anemia, hypoxia, or a combination of all of the above. Aortic Stenosis f.
Interpretation: There is sinus tachycardia, with right bundle branch block (RBBB). Blunt cardiac injury my result in : 1) Acute myocardial rupture with tamponade 2) Valve rupture (tricuspid, aortic, mitral) 3) Coronary thrombosis or dissection (and thus Acute MI) from direct coronary blunt injury 4) Dysrhythmias of all kinds.
2 cases of Aortic Stenosis: Diffuse Subendocardial Ischemia on the ECG. Systematic Assessment of the ECG in Figure-1: My Descriptive Analysis of ECG findings in Figure-1 is as follows: Sinus tachycardia at ~110/minute. A slightly prolonged QTc ( although this is difficult to assess given the tachycardia ). Left main?
The patient had no hypertension, no tachycardia, a normal hemoglobin, no drug use, no hypotension/shock, no murmur of aortic stenosis. We also looked at his aortic root by both parasternal and suprasternal views, and the aorta was normal.]
Other than tachycardia, Other than slight tachycardia, vitals were within normal limits (including oxygen saturation). Chest pain and Concordant ST Depression in a patient with aortic valve and previously normal angiogram Right Bundle Branch Block and ST Depression in V1-V3. Apparently he denied chest pain. Is that normal?
No signs for aortic dissection or pulmonary embolus. --"Results were discussed with the ordering physician. That said, against acute PE — is the inconstancy of this patient's symptoms — the lack of tachycardia — and the lack of any other ECG signs of acute RV strain. A CT Coronary angiogram was ordered. CAD-RADS category 1. --No
Sinus Tachycardia ( common in any trauma patient. ). Other Arrhythmias ( PACs, PVCs, AFib, Bradycardia and AV conduction disorders — potentially lethal VT/VFib ). RBBB ( as by far the most common conduction defect — owing to the more vulnerable anatomic location of the RV ).
Here is another proven left main occlusion in a young woman who presented with sudden pulmonary edema, had this ECG recorded, then arrested and was resuscitated after 30 minutes of CPR: This has sinus tachycardia with RBBB and LAFB, and STE in V2-V6 as well as I, aVL This pattern could just as easily be seen in LAD occlusion. Knotts et al.
Aortic Dissection, Valvular (especially Aortic Stenosis), Tamponade. If the patient has Abnormal Vital Signs (fever, hypotension, tachycardia, or tachypnea, or hypoxemia), then these are the primary issue to address, as there is ongoing pathology which must be identified. heart auscultation (aortic stenosis); c.
The status of the patients chest pain at this time is unknown : EKG 1, 1300: There is sinus tachycardia and artifact of low and high frequency. However, there is also significant tachycardia , with heart rate of 116, and known hypoxia. Smith : "decompensation" of aortic stenosis might have initiated this entire cascade.
Important: It is exceedingly rare for an anterior STEMI to be due to Aortic Dissection. Here it is: Type A Aortic Dissection Why was the troponin so elevated? The CT showed extensive type A aortic dissection which starts at the ostium of the RCA and extends all the way to the left iliac artery. This is easy to see.
Mind you, even an innocuous episode of fever, associated dyspnea, and tachycardia can elevate the mitral gradient and sound a false alarm. It needs a good knowledge of anatomy, physiology of inter & Intra valvular hemodynamics.It Depending solely on prosthetic valve gradients to diagnose obstruction is the biggest error we commit.
The QoH now correctly sees active OMI: A CT scan to rule out aortic dissection was performed (Smith: why???) Hans has not even been to medical school. One's training has NOTHING to do with one's OMI ECG interpretation skills. before transferring the patient to the cath facility. It was without evidence of dissection.
Same story for pressure half time, especially with tachycardia and little mitral regurgitation. There is something called low gradient severe MS (as in aortic stenosis). After all, we all know, with years of experience in echocardiography ,in a funnel-shaped degenerated mitral valve, we can get whatever MVO we desire to report !
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