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The provider had sent the patient for an aortic dissection scan which had shown extremely heavy calcification of the LAD. There was a 100% proximal LAD occlusion that was opened and stented. The cath lab was activated. But 45 minutes later than it should have been.
The Queen of Hearts disagrees, diagnosing OMI with high confidence: Case Continued: The EKG was not immediately recognized by the emergency provider, who ordered a CT scan to rule out aortic dissection at 1419. This was a presumed culprit and a stent was placed. Assuming that was indeed a culprit, then this was ACS.
As his pain was very severe, emergency physicians concerned of aortic dissection and ordered a thoracic CT scan. The lesion was successfully stented. Take home messages: 1- In STEMI/NSTEMI paradigm you search for STE on ECG. Bi-phasic scan showed no dissection or pulmonary embolism. Turk Kardiyol Dern Ars. doi: 10.5543/tkda.2021.21026.
The "criteria" for posterior STEMI are 0.5 Is it STEMI or NonSTEMI? The patient had no hypertension, no tachycardia, a normal hemoglobin, no drug use, no hypotension/shock, no murmur of aortic stenosis. We also looked at his aortic root by both parasternal and suprasternal views, and the aorta was normal.]
Step 1 to missing posterior MI is relying on the STEMI criteria. A prospective validation of STEMI criteria based on the first ED ECG found it was only 21% sensitive for Occlusion MI, and disproportionately missed inferoposterior OMI.[1] But it is still STEMI negative. A 15 lead ECG was done (below).
A middle-aged male with h/o CAD and stents presented with typical chest pressure. It may be difficult to read STEMI in the setting of RBBB. There is, however, a long QT also, with abnormal T-waves, but this is not STEMI. So there is pathologic ST elevation here, consistent with anterolateral STEMI. What do you think?
When total LM occlusion does present with STE in aVR, there is ALWAYS ST Elevation elsewhere which makes STEMI obvious; in other words, STE is never limited to only aVR but instead it is part of a massive and usually obvious STEMI. All are, however, clearly massive STEMI. This is her ECG: An obvious STEMI, but which artery?
A repeat ECG was done: Obvious anterolateral wall STEMI. On arrival his BP was 70s/40s, so an intra-aortic ballon pump was placed. The patient was taken back to the cath lab, where 100% proximal in-stent rethrombosis was found and treated. This rhythm reportedly produced no palpable pulse, and CPR was continued.
There is ventricular hypertrophy in the absence of abnormal loading conditions, such as aortic stenosis, or hypertension, for example – of which the most common variant is Asymmetric Septal Hypertrophy. This worried the crew of potential acute coronary syndrome and STEMI was activated pre-hospital.
No signs for aortic dissection or pulmonary embolus. --"Results were discussed with the ordering physician. INTERVENTION * Successful angioplasty and stenting (drug eluting) of the mid LAD * Successful angioplasty of the ostial 1st diagonal Learning points: 1. Transient STEMI is at high risk of re-occlusion.
His first EKG is shown below, with a lead II rhythm strip: EKG 1, 1645 A provisder who is looking for STEMI would not see much in this EKG. It is possible that the T waves in this EKG are of an intermediate morphology between full-blown STEMI and inferior reperfusion. The thrombus was aspirated and the distal RCA was stented.
At cath later the same day, a proximal 99% RCA culprit lesion was stented. That said — the overall picture to me did not "look" acute — and the history of "chest pain radiating to the back " in this 80-something man with marked LVH — made me strongly consider an aortic dissection as a more likely cause. Troponin T peaked at 4051 ng/L.
His initial high sensitivity troponin I returned at 1300 ng/L and given that his cardiac workup was otherwise unremarkable, a CT was obtained to evaluate for pulmonary embolism and aortic aneurysm or dissection but this too was unrevealing. Another EKG was also obtained. ECG at time 82 minutes: What do you think?
It is diagnostic of OMI, but this is SUBACUTE OMI I sent this ECG to my "EKG Nerdz" friends, without any clinical info at all and they answered "OMI" The Queen said: "STEMI-Equivalent with High Confidence:" Notice she sees findings in both normal beats and PVCs. It was opened and stented. If this were ACUTE (vs.
The cath lab was deactivated by cardiologist on arrival at ED because it was "not a STEMI". No thoracic aortic hematoma, aneurysm or dissection. First obtuse marginal also had an 80% stenosis and was stented. Pt received 324 ASA and 2 sprays of nitro with improvement. Cath lab was activated by EMS and transported emergent."
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