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Look at the aortic outflow tract. The diagnostic coronary angiogram identified only minimal coronary artery disease, but there was a severely calcified, ‘immobile’ aortic valve. Aortic angiogram did not reveal aortic dissection. What do you see? Answer below in the still shot.
Two weeks ago he had a significant MVC with many severe injuries, including aortic injury s/p endovascular repair. Serial echo monitoring showed increasing pericardial pressures without overt tamponade physiology. That said — I did not interpret these differences as the result of acute ischemia.
The first troponin returned at 0.099 ng/mL (elevated, consistent with Non-Occlusion MI) Providers were concerned with aortic dissection, so they order a chest aorta CT. This transmural ischemia, but not necessarily completed infarction (yet).
Written by Pendell Meyers A woman in her 20s with connective tissue disorder and history of aortic root and valve repair presented with palpitations. Further history revealed she had new onset atrial flutter soon after her aortic surgery, and was put on flecainide approximately 1 month ago. Here is her triage ECG: What do you think?
But it need not imply the actual functional significance of the stenosis in terms of flow physiology. FFR is obtained by dividing the pressure distal to the stenosis by the central aortic pressure, which is usually equal to the pressure proximal to the stenosis if there is no additional stenosis in between. Normal FFR is 1.0
Aortic Dissection, Valvular (especially Aortic Stenosis), Tamponade. Evidence of acute ischemia (may be subtle) vii. heart auscultation (aortic stenosis); c. 2nd or 3rd degree AV blocks or sinus pause of at least 2 seconds iv. Right bundle branch block (BBB) with hemiblock (bifascicular block) v. Left BBB vi. LVH or RV d.
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