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The Queen of Hearts disagrees, diagnosing OMI with high confidence: Case Continued: The EKG was not immediately recognized by the emergency provider, who ordered a CT scan to rule out aortic dissection at 1419. Smith comment 2: I frequently see failure to control BP in patients with acute chest pain or acute heartfailure.
Look at the aortic outflow tract. The diagnostic coronaryangiogram identified only minimal coronary artery disease, but there was a severely calcified, ‘immobile’ aortic valve. Aorticangiogram did not reveal aortic dissection. What do you see? Answer below in the still shot.
There is ventricular hypertrophy in the absence of abnormal loading conditions, such as aortic stenosis, or hypertension, for example – of which the most common variant is Asymmetric Septal Hypertrophy. Type II MI), however decided to pursue coronaryangiogram out of an abundance of caution.
While the first one may radiate to the axilla and base, but usually not into the neck, it does reflect both aortic outflow obstruction and mitral regurgitation in patients with a large gradient. On the other hand, the murmur in valvular aortic stenosis does not change substantially or decreases slightly following the Valsalva maneuver.
CT angiogram chest: no aortic dissection or pulmonary embolism. He was transitioned to oral heartfailure medications and discharged home slightly over one week after presentation. He was readmitted a few weeks later for a heartfailure exacerbation, diuresed, and discharged again.
Anything that causes pulmonary edema: poor LV function, fluid overload, previous heartfailure (HFrEF or HFpEF), valvular disease. Smith : "decompensation" of aortic stenosis might have initiated this entire cascade. What "initiates" the aortic stenosis cascade? What other pathology is possible?
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