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A comparison of electrocardiographic changes during reperfusion of acute myocardial infarction by thrombolysis or percutaneous transluminal coronary angioplasty. Two ECG patterns were described by the original investigators in 1982 as being consistent with a Wellens’ Syndrome prediction of high-grade LAD stenosis. Am Heart J.
Rescue treatment with stenting, balloon angioplasty, and/or intraarterial thrombolysis or antiplatelets are often required to treat the underlying stenosis. IntroductionIntracranial atherosclerotic disease (ICAD) is associated with up to 32% of posterior circulation strokes.1
Angiogram --LAD is a large-caliber vessel that wraps around the apex --There is a tubular 80% in the ostial LAD that was relatively smooth but did not resolve with repeated doses of IC nitroglycerin --There is a tiny D1, medium D2, and small D3 Lesion on Prox LAD: Ostial 80% stenosis. Pre- procedure TIMI III flow was noted. Am Heart J.
Characteristic electrocardiographic pattern indicating a critical stenosis high in left anterior descending coronary artery in patients admitted because of impending myocardial infarction. Am Heart J (1989) 117 : pp 657-665. de Zwaan C., Wellens H.J.J., Am Heart J (1982) 103 : pp 730-736. Doevendans P.A., Gorgels A.P., van der Zee R.,
Dormu performed an aortogram of the bilateral lower extremity with bilateral iliac runoff, which revealed a 90% stenosis of the right superficial femoral artery and 100% occlusion of all three tibial vessels. These studies revealed an 80% stenosis of the left superficial femoral artery and 100% occlusion of all three tibial vessels.
Characteristic electrocardiographic pattern indicating a critical stenosis high in left anterior descending coronary artery in patients admitted because of impending myocardial infarction. Wellens' original Papers : de Zwaan C, Bär FW, Wellens HJ. Am Heart J. 1982 Apr;103(4 Pt 2):730-6. de Zwaan C et al. Am Heart J. 2000 Mar;139(3):430-6.
Cath report later that afternoon, around 25 hours since arrival: "There was a 70% proximal LAD stenosis secondary to a spontaneous coronary dissection with narrowing of the proximal LAD to at least 50% stenosis. Severe hypokinesis of the anteroseptal, anterior, inferoseptal, and apical myocardium. Normal RV function.
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