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This is a value typical for a large subacute MI, n ormal value 48 hours after myocardialinfarction is associated with Post-Infarction Regional Pericarditis ( PIRP ). As already mentioned, this patient could have post-infarction regional pericarditis from a large completed MI.
A middle-aged woman had intermittent angina for 48 hours, then onset of constant, crushing chest pain for 1.5 Appearance of abnormal Q waves early in the course of acute myocardialinfarction: implications for efficacy of thrombolytic therapy. of AMI patients and is often preceded by postinfarction regional pericarditis (PIRP).
I do not think this is acute occlusion myocardialinfarction (OMI). OMI is generally of more acute onset, unless there is intermittent angina. There is also mild pericardial enhancement consistent with pericarditis. Get an emergent contrast echocardiogram. These are reasons why it does not look like OMI: 1.
So in anterior leads, for diagnosis of ST elevation myocardialinfarction, V1, the cutoff is usually 2 mm, while 1 mm is enought in other leads. That is usually with angina and ventricular strain patterns. PR segment elevation and depression can occur in atrial infarction and pericarditis.
The patient might be having cardiac ischemia, but if he is, it is unstable angina or non-STEMI, or perhaps he has not YET pseudonormalized, so serial ECGs may be important. Differential of peri-infarct pericardial fluid The differential includes 1) pericarditis with effusion or 2) hemopericardium.
The exception is with postinfarction pericarditis , in which a completed transmural infarct results in inflammation of the subepicardial myocardium and STE in the distribution of the infarct, and which results in increased STE and large upright T-waves. These findings together are more commonly seen with pericarditis.
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