Remove Angina Remove Arrhythmia Remove STEMI
article thumbnail

Management and outcomes of spontaneous coronary artery dissection: a systematic review of the literature

Frontiers in Cardiovascular Medicine

with ST elevated myocardial infarction (STEMI), 3.41% with unstable angina, 0.56% with stable angina, and 0.11% were diagnosed with various types of arrhythmias. Approximately 48.5% of the patients were diagnosed with non-ST elevated myocardial infarction (NSTEMI), 36.8% There were initially 65.2%

SCAD 75
article thumbnail

46 year old with chest pain develops a wide complex rhythm -- see many examples

Dr. Smith's ECG Blog

There are three mechanisms of arrhythmia: automatic, re-entry, and triggered. The most common triggered arrhythmia is Torsades de Pointes. It is a benign arrhythmia which requires no specific treatment. The receiving emergency physician consulted with interventional cardiology who stated there was no STEMI. Moffat, M.

article thumbnail

Is this a STEMI? No, not by definition! Why not? Why is this Important?

Dr. Smith's ECG Blog

This is all but diagnostic of STEMI, probably due to wraparound LAD The cath lab was activated. Therefore this is " Transient ST Elevation Unstable Angina." As there was ruptured plaque, this is NOT Prinzmetal's angina. Here are many other cases of Unstable Angina , in spite of Eugene Braunwald's Requiem for Unstable Angina.

STEMI 52
article thumbnail

Three normal high sensitivity troponins over 4 hours with a "normal ECG"

Dr. Smith's ECG Blog

Thus, the patient does not (yet) get a formal diagnosis of MI and must be called unstable angina unless further troponins return above the 99th percentile. On the basis of unresolved angina, cardiology decided to perform rescue PCI. Medically refractory angina should have immediate angiography, but this only happens 6.4%

Angina 107
article thumbnail

Precordial ST depression. What is the diagnosis?

Dr. Smith's ECG Blog

A middle aged male with no h/o CAD presented with one week of crescendo exertional angina, and had chest pain at the time of the first ECG: Here is the patient's previous ECG: Here is the patient's presenting ED ECG: There is isolated ST depression in precordial leads, deeper in V2 - V4 than in V5 or V6. There is no ST elevation.

STEMI 52
article thumbnail

ECG Blog #386 — OMI or Something Else?

Ken Grauer, MD

The finding of all negative QRS complexes in leads V3-thru- V6 therefore strongly suggests that the arrhythmia-associated impulse is not traveling over an AP ( Steurer et al — Clin. This should result in at least some positivity of QRS complexes as one moves toward the lateral chest leads. Cardiol 17:306-308, 1994 ).

Blog 78
article thumbnail

Why many Ischemic VTs are not abolished by revascularization ?

Dr. S. Venkatesan MD

Relationship between Ischemia and arrhythmia While the relationship between Ischemia and VT is really complex, the term “Ischemic VT” itself adds more twists. In chronic VT , shooting down the ischemic focus by ablation its more likely to extinguish the arrhythmia ,rather than revascularization.