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She had a single chamber ICD/Pacemaker implanted several years prior due to ventricular tachycardia. Answer : The ECG above shows a regular wide complex tachycardia. Said differently, the ECG shows a rather slow ventricular tachycardia with a 2:1 VA conduction. Cardiac output (CO) was being maintained by the tachycardia.
The best approach for ablating ventricular tachycardia (VT) targeting right ventricular free wall (RVFW) aneurysms in arrhythmogenic right ventricular cardiomyopathy (ARVC) remains undefined.
LV aneurysm has QS-waves, so this couldn't be LV aneurysm, right? RBBB makes it mandatory that there are R'-waves even in the presence of LV aneurysm. Additionally, it is very difficult to differentiate subacute reperfused OMI from LV aneurysm: both have Q-waves and inverted T-waves. This is HIGHLY suspicious for OMI.
ABSTRACT Introduction Left atrial appendage aneurysm (LAAA) is a rare congenital cardiac anomaly that involves the progressive dilatation of the left atrial appendage (LAA), predisposing the patient to serious complications such as atrial tachyarrhythmias, life-threatening systemic thromboembolism, and cardiac dysfunction.
He was rushed by residents into our critical care room with a diagnosis of STEMI, and they handed me this ECG: There is sinus tachycardia with ST elevation in II, III, and aVF, as well as V4-V6. ACS and STEMI generally do not cause tachycardia unless there is cardiogenic shock. He had this ECG recorded. Are the lungs clear?
His previous echo one month prior shows the same thing: “consistent with old infarct in LAD vascular territory, with EF 45%” "I think there is something else causing his tachycardia which is exaggerating his EKG findings and mimicking an acute myocardial infarction." The patient spontaneously converted back to sinus tachycardia.
4 We hereby reported a case of atrial tachycardia (AT)/AF originating from an aneurysmal interatrial septum (AIS) that was successfully mapped with a high-density mapping catheter and eliminated with pulsed field ablation (PFA). Extra-pulmonary vein (PV) triggers have been reported in up to 4.9-15%
Here is his ED ECG: There is sinus tachycardia. The amount of ST elevation and depression is slightly less than on the ECG above, but there is also no tachycardia, which tends to exaggerate ST deviation. First, to be called "aneurysm," the MI must be at least a couple weeks old, so this case does not strictly apply.
Smith : Old inferior MI with persistent ST Elevation ("inferior aneurysm") has well-formed Q-waves. In inferior aneurysm, we usually see QR-waves, whereas for anterior aneurysm, we see QS-waves (no R- or r-wave at all!). The patient had a history of ‘NSTEMI’ a decade prior, with an RCA stent.
In that study commonest ECG abnormalites were QTc prolongation followed by brady/tachycardia and then ST segment deviations [3]. Sometimes cardiac troponin elevation may accompany ECG changes in aneurysmal subarachnoid hemorrhage. But the number of persons with lobar hemorrhage in that study was only 17%. 2009 Nov;40(11):3478-84.
Peak troponin was a massive 500,000 ng/L, echo showed EF reduced to 20%, and follow up ECG showed LV aneurysm morphology with anterior Q wave and persisting ST elevation. There is sinus tachycardia at ~100/minute. In today's case — the sinus tachycardia may have been a harbinger of this patient's ultimate demise. As per Dr.
Prior ECG available on file from 2 months before: We do not know the clinical events happening during this ECG, but there is borderline tachycardia, PVCs, and likely some evidence of subendocardial ischemia with small STDs maximal in V5-6/II, slight reciprocal STE in aVR. QS waves from V2-V5 consistent with LV aneurysm morphology.
The EKG is diagnostic of acute inferior, posterior, and lateral OMI superimposed on “LV aneurysm” morphology. He had multiple episodes of bradycardia and nonsustained ventricular tachycardia. Patient 2 , EKG 1: What do you think? Patient 1 ’s EKG was obtained first, so it was interpreted first. It was stented.
The only time you see this without ischemia is when there is an abnormal QRS, such as LVH, LBBB, LV aneurysm (old MI with persistent STE) or WPW." In such cases, it is common for tachycardia to exaggerate the ST Elevation And, in fact, there was no new acute MI at this visit - troponins did not rise again. Troponin I greater than 1.0
In addition to sinus tachycardia, the only abnormalities listed by the computer were "low voltage, precordial leads" and "anteroseptal infarct, old.Q Repeat ECG at 1624 (shortly before cath): QS waves now present in V2-V3, with slight STE, showing the pattern of left ventricular aneurysm morphology. Case 2: What do you think?
There was near transmural late gadolinium enhancement (LGE) of the aneurysm and an associated 7 mm of MAD with posterior mitral valve prolapse (MVP). The scar and aneurysm that develop can serve as substrate for reentrant VA. Further work up included cardiac magnetic resonance which identified a 4 x 1.8
The status of the patients chest pain at this time is unknown : EKG 1, 1300: There is sinus tachycardia and artifact of low and high frequency. There are no Q-waves to suggest old inferior MI, or inferior aneurysm as the etiology of the ST Elevation. She arrived to the ED with a nonrebreather mask. An EKG was immediately recorded.
Look for Vascular Etiology -- think of these while doing H and P: --Bleeding: ruptured AAA, GI bleed, ruptured ectopic pregnancy, other spontaneous bleed such as mesenteric aneurysms. Most physicians will automatically be worried about these symptoms. The tracings were considered abnormal in the following cases: 1.
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