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Ken (below) is appropriately worried about pulmonaryembolism from the ECG. What I had not told him before he made that judgement is that the patient also had ultrasound B-lines of pulmonary edema. LV aneurysm has QS-waves, so this couldn't be LV aneurysm, right? What do you think?
Smith comment: before reading anything else, this case screamed pulmonaryembolism to me. I would do bedside ultrasound to look at the RV, look for B lines as a cause of hypoxia (which would support OMI, and argue against PE), and if any doubt persists, a rapid CT pulmonary angiogram.
There are two forms of HHT: (HHT1) has a higher incidence in women and typically involves pulmonary and cerebral arteriovenous malformations (AVM). Given the heavy cerebral AVM burden with multiple cerebral artery feeders, the AVM angiosclerosis and basilar artery aneurysmembolization were deferred.
IntroductionSubarachnoid Hemorrhage (SAH) resulting from the spontaneous rupture of an aneurysm is a rare and highly debilitating condition. Despite its severity, patients with aneurysmal SAH remain understudied, particularly concerning the evaluation of the incidence and consequences of subsequent acute kidney injury (AKI).
The patient's heart had significant recovery: Echo : Estimated LVEF 32%, apical wall motion abnormality with diastolic distortion (LV aneurysm), suggestive of old MI. pulmonaryembolism, sepsis, etc.), Coronary thrombosis or embolism can result in MINOCA, either with or without a hypercoagulable state. myocarditis).
CT angiogram chest: no aortic dissection or pulmonaryembolism. Serial chest xrays: progressive bilateral pulmonary edema. Repeat CT angio chest (not CT coronary, unclear what protocol) showed possible LAD aneurysm and thrombus. No further troponins were measured. No further cath details available.
Look for Vascular Etiology -- think of these while doing H and P: --Bleeding: ruptured AAA, GI bleed, ruptured ectopic pregnancy, other spontaneous bleed such as mesenteric aneurysms. Serious outcomes included death, arrhythmia, myocardial infarction, structural heart disease, pulmonaryembolism, and hemorrhage. of ED visits.
His initial high sensitivity troponin I returned at 1300 ng/L and given that his cardiac workup was otherwise unremarkable, a CT was obtained to evaluate for pulmonaryembolism and aortic aneurysm or dissection but this too was unrevealing. Also: electrical instability, pulmonary edema, or hypotension.
No thoracic aortic hematoma, aneurysm or dissection. No pulmonaryembolism is identified. While in the ED, patient's pain worsened to previous severity of 6/10 pain and improved to 3/10 on NTG drip. CT Angio Chest IMPRESSION 1. There are moderate coronary artery calcifications.
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